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Items: 1 to 20 of 127

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Fine-mapping loss of gene architecture at the CDKN2B (p15INK4b), CDKN2A (p14ARF, p16INK4a), and MTAP genes in head and neck squamous cell carcinoma.

Worsham MJ, Chen KM, Tiwari N, Pals G, Schouten JP, Sethi S, Benninger MS.

Arch Otolaryngol Head Neck Surg. 2006 Apr;132(4):409-15.

PMID:
16618910
6.

Methylthioadenosine phosphorylase regulates ornithine decarboxylase by production of downstream metabolites.

Subhi AL, Diegelman P, Porter CW, Tang B, Lu ZJ, Markham GD, Kruger WD.

J Biol Chem. 2003 Dec 12;278(50):49868-73.

7.

Concordant loss of MTAP and p16/CDKN2A expression in gastroesophageal carcinogenesis: evidence of homozygous deletion in esophageal noninvasive precursor lesions and therapeutic implications.

Powell EL, Leoni LM, Canto MI, Forastiere AA, Iocobuzio-Donahue CA, Wang JS, Maitra A, Montgomery E.

Am J Surg Pathol. 2005 Nov;29(11):1497-504.

PMID:
16224217
8.

Methylthioadenosine phosphorylase as target for chemoselective treatment of T-cell acute lymphoblastic leukemic cells.

Efferth T, Miyachi H, Drexler HG, Gebhart E.

Blood Cells Mol Dis. 2002 Jan-Feb;28(1):47-56.

PMID:
11987241
9.

Homozygous deletions of methylthioadenosine phosphorylase in human biliary tract cancers.

Karikari CA, Mullendore M, Eshleman JR, Argani P, Leoni LM, Chattopadhyay S, Hidalgo M, Maitra A.

Mol Cancer Ther. 2005 Dec;4(12):1860-6.

10.

Methylthioadenosine phosphorylase gene deletions are common in osteosarcoma.

García-Castellano JM, Villanueva A, Healey JH, Sowers R, Cordon-Cardo C, Huvos A, Bertino JR, Meyers P, Gorlick R.

Clin Cancer Res. 2002 Mar;8(3):782-7.

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Strong expression of methylthioadenosine phosphorylase (MTAP) in human colon carcinoma cells is regulated by TCF1/[beta]-catenin.

Bataille F, Rogler G, Modes K, Poser I, Schuierer M, Dietmaier W, Ruemmele P, Mühlbauer M, Wallner S, Hellerbrand C, Bosserhoff AK.

Lab Invest. 2005 Jan;85(1):124-36.

15.

Promoter-hypermethylation is causing functional relevant downregulation of methylthioadenosine phosphorylase (MTAP) expression in hepatocellular carcinoma.

Hellerbrand C, Mühlbauer M, Wallner S, Schuierer M, Behrmann I, Bataille F, Weiss T, Schölmerich J, Bosserhoff AK.

Carcinogenesis. 2006 Jan;27(1):64-72.

16.

A methylthioadenosine phosphorylase (MTAP) fusion transcript identifies a new gene on chromosome 9p21 that is frequently deleted in cancer.

Schmid M, Sen M, Rosenbach MD, Carrera CJ, Friedman H, Carson DA.

Oncogene. 2000 Nov 23;19(50):5747-54.

18.

Concordant loss of MTAP and p16/CDKN2A expression in pancreatic intraepithelial neoplasia: evidence of homozygous deletion in a noninvasive precursor lesion.

Hustinx SR, Leoni LM, Yeo CJ, Brown PN, Goggins M, Kern SE, Hruban RH, Maitra A.

Mod Pathol. 2005 Jul;18(7):959-63.

19.

Methylthioadenosine phosphorylase cDNA transfection alters sensitivity to depletion of purine and methionine in A549 lung cancer cells.

Hori H, Tran P, Carrera CJ, Hori Y, Rosenbach MD, Carson DA, Nobori T.

Cancer Res. 1996 Dec 15;56(24):5653-8.

20.

CDKN2A, CDKN2B, and MTAP gene dosage permits precise characterization of mono- and bi-allelic 9p21 deletions in childhood acute lymphoblastic leukemia.

Bertin R, Acquaviva C, Mirebeau D, Guidal-Giroux C, Vilmer E, Cavé H.

Genes Chromosomes Cancer. 2003 May;37(1):44-57.

PMID:
12661005
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