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Items: 1 to 20 of 216

1.
3.

Toxicity of familial ALS-linked SOD1 mutants from selective recruitment to spinal mitochondria.

Liu J, Lillo C, Jonsson PA, Vande Velde C, Ward CM, Miller TM, Subramaniam JR, Rothstein JD, Marklund S, Andersen PM, Brännström T, Gredal O, Wong PC, Williams DS, Cleveland DW.

Neuron. 2004 Jul 8;43(1):5-17.

4.

CuZn superoxide dismutase (SOD1) accumulates in vacuolated mitochondria in transgenic mice expressing amyotrophic lateral sclerosis-linked SOD1 mutations.

Jaarsma D, Rognoni F, van Duijn W, Verspaget HW, Haasdijk ED, Holstege JC.

Acta Neuropathol. 2001 Oct;102(4):293-305.

PMID:
11603803
5.

Mutant SOD1 in neuronal mitochondria causes toxicity and mitochondrial dynamics abnormalities.

Magrané J, Hervias I, Henning MS, Damiano M, Kawamata H, Manfredi G.

Hum Mol Genet. 2009 Dec 1;18(23):4552-64. doi: 10.1093/hmg/ddp421. Epub 2009 Sep 24.

6.

Early vacuolization and mitochondrial damage in motor neurons of FALS mice are not associated with apoptosis or with changes in cytochrome oxidase histochemical reactivity.

Bendotti C, Calvaresi N, Chiveri L, Prelle A, Moggio M, Braga M, Silani V, De Biasi S.

J Neurol Sci. 2001 Oct 15;191(1-2):25-33.

PMID:
11676989
7.

Mutant SOD1 linked to familial amyotrophic lateral sclerosis, but not wild-type SOD1, induces ER stress in COS7 cells and transgenic mice.

Tobisawa S, Hozumi Y, Arawaka S, Koyama S, Wada M, Nagai M, Aoki M, Itoyama Y, Goto K, Kato T.

Biochem Biophys Res Commun. 2003 Apr 4;303(2):496-503.

PMID:
12659845
8.

The neuroprotective factor Wlds does not attenuate mutant SOD1-mediated motor neuron disease.

Vande Velde C, Garcia ML, Yin X, Trapp BD, Cleveland DW.

Neuromolecular Med. 2004;5(3):193-203.

PMID:
15626820
9.

Cytochrome c association with the inner mitochondrial membrane is impaired in the CNS of G93A-SOD1 mice.

Kirkinezos IG, Bacman SR, Hernandez D, Oca-Cossio J, Arias LJ, Perez-Pinzon MA, Bradley WG, Moraes CT.

J Neurosci. 2005 Jan 5;25(1):164-72.

10.

Mouse motor neuron disease caused by truncated SOD1 with or without C-terminal modification.

Watanabe Y, Yasui K, Nakano T, Doi K, Fukada Y, Kitayama M, Ishimoto M, Kurihara S, Kawashima M, Fukuda H, Adachi Y, Inoue T, Nakashima K.

Brain Res Mol Brain Res. 2005 Apr 27;135(1-2):12-20.

PMID:
15857664
12.

Mutant SOD1G93A triggers mitochondrial fragmentation in spinal cord motor neurons: neuroprotection by SIRT3 and PGC-1α.

Song W, Song Y, Kincaid B, Bossy B, Bossy-Wetzel E.

Neurobiol Dis. 2013 Mar;51:72-81. doi: 10.1016/j.nbd.2012.07.004. Epub 2012 Jul 20.

13.

Ultrastructural study of mitochondria in the spinal cord of transgenic mice with a G93A mutant SOD1 gene.

Sasaki S, Warita H, Murakami T, Abe K, Iwata M.

Acta Neuropathol. 2004 May;107(5):461-74. Epub 2004 Mar 17.

PMID:
15029445
14.
15.

Calcium-permeable AMPA receptors promote misfolding of mutant SOD1 protein and development of amyotrophic lateral sclerosis in a transgenic mouse model.

Tateno M, Sadakata H, Tanaka M, Itohara S, Shin RM, Miura M, Masuda M, Aosaki T, Urushitani M, Misawa H, Takahashi R.

Hum Mol Genet. 2004 Oct 1;13(19):2183-96. Epub 2004 Aug 4.

PMID:
15294873
16.
17.

Restricted expression of G86R Cu/Zn superoxide dismutase in astrocytes results in astrocytosis but does not cause motoneuron degeneration.

Gong YH, Parsadanian AS, Andreeva A, Snider WD, Elliott JL.

J Neurosci. 2000 Jan 15;20(2):660-5.

18.

In vivo pathogenic role of mutant SOD1 localized in the mitochondrial intermembrane space.

Igoudjil A, Magrané J, Fischer LR, Kim HJ, Hervias I, Dumont M, Cortez C, Glass JD, Starkov AA, Manfredi G.

J Neurosci. 2011 Nov 2;31(44):15826-37. doi: 10.1523/JNEUROSCI.1965-11.2011.

19.

Overexpression of CCS in G93A-SOD1 mice leads to accelerated neurological deficits with severe mitochondrial pathology.

Son M, Puttaparthi K, Kawamata H, Rajendran B, Boyer PJ, Manfredi G, Elliott JL.

Proc Natl Acad Sci U S A. 2007 Apr 3;104(14):6072-7. Epub 2007 Mar 26.

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