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Items: 1 to 20 of 87

1.

Evidence of a role for nonmuscle myosin II in herpes simplex virus type 1 egress.

van Leeuwen H, Elliott G, O'Hare P.

J Virol. 2002 Apr;76(7):3471-81.

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Herpes simplex virus 1 VP22 regulates translocation of multiple viral and cellular proteins and promotes neurovirulence.

Tanaka M, Kato A, Satoh Y, Ide T, Sagou K, Kimura K, Hasegawa H, Kawaguchi Y.

J Virol. 2012 May;86(9):5264-77. doi: 10.1128/JVI.06913-11. Epub 2012 Feb 22.

5.

UL20 protein functions precede and are required for the UL11 functions of herpes simplex virus type 1 cytoplasmic virion envelopment.

Fulmer PA, Melancon JM, Baines JD, Kousoulas KG.

J Virol. 2007 Apr;81(7):3097-108. Epub 2007 Jan 10.

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The major tegument structural protein VP22 targets areas of dispersed nucleolin and marginalized chromatin during productive herpes simplex virus 1 infection.

López MR, Schlegel EF, Wintersteller S, Blaho JA.

Virus Res. 2008 Sep;136(1-2):175-88. doi: 10.1016/j.virusres.2008.05.010. Epub 2008 Jun 12.

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Non-muscle myosin IIA is a functional entry receptor for herpes simplex virus-1.

Arii J, Goto H, Suenaga T, Oyama M, Kozuka-Hata H, Imai T, Minowa A, Akashi H, Arase H, Kawaoka Y, Kawaguchi Y.

Nature. 2010 Oct 14;467(7317):859-62. doi: 10.1038/nature09420.

PMID:
20944748
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A conserved region of the herpes simplex virus type 1 tegument protein VP22 facilitates interaction with the cytoplasmic tail of glycoprotein E (gE).

O'Regan KJ, Bucks MA, Murphy MA, Wills JW, Courtney RJ.

Virology. 2007 Feb 5;358(1):192-200. Epub 2006 Sep 25.

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A network of protein interactions around the herpes simplex virus tegument protein VP22.

Maringer K, Stylianou J, Elliott G.

J Virol. 2012 Dec;86(23):12971-82. doi: 10.1128/JVI.01913-12. Epub 2012 Sep 19.

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