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Items: 1 to 20 of 120

1.

A comprehensive review of genetic association studies.

Hirschhorn JN, Lohmueller K, Byrne E, Hirschhorn K.

Genet Med. 2002 Mar-Apr;4(2):45-61. Review.

PMID:
11882781
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Meta-analysis of genetic association studies supports a contribution of common variants to susceptibility to common disease.

Lohmueller KE, Pearce CL, Pike M, Lander ES, Hirschhorn JN.

Nat Genet. 2003 Feb;33(2):177-82. Epub 2003 Jan 13.

PMID:
12524541
6.

What can genome-wide association studies tell us about the genetics of common disease?

Iles MM.

PLoS Genet. 2008 Feb;4(2):e33. doi: 10.1371/journal.pgen.0040033.

7.

Isolates and their potential use in complex gene mapping efforts.

Varilo T, Peltonen L.

Curr Opin Genet Dev. 2004 Jun;14(3):316-23. Review.

PMID:
15172676
8.

On the advantage of haplotype analysis in the presence of multiple disease susceptibility alleles.

Morris RW, Kaplan NL.

Genet Epidemiol. 2002 Oct;23(3):221-33.

PMID:
12384975
9.

Finding genes influencing susceptibility to complex diseases in the post-genome era.

Rannala B.

Am J Pharmacogenomics. 2001;1(3):203-21. Review.

PMID:
12083968
10.

Genetic association studies of complex traits: design and analysis issues.

Newton-Cheh C, Hirschhorn JN.

Mutat Res. 2005 Jun 3;573(1-2):54-69. Review.

PMID:
15829237
11.

Mining for SNPs: putting the common variants--common disease hypothesis to the test.

Cargill M, Daley GQ.

Pharmacogenomics. 2000 Feb;1(1):27-37. Review.

PMID:
11258594
12.

Significance levels in genome scans.

Thomson G.

Adv Genet. 2001;42:475-86. Review.

PMID:
11037336
13.

Mapping the new frontier: complex genetic disorders.

Mayeux R.

J Clin Invest. 2005 Jun;115(6):1404-7. Review.

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Common deletions and SNPs are in linkage disequilibrium in the human genome.

Hinds DA, Kloek AP, Jen M, Chen X, Frazer KA.

Nat Genet. 2006 Jan;38(1):82-5. Epub 2005 Dec 4.

PMID:
16327809
16.

What genome-wide association studies can do for medicine.

Christensen K, Murray JC.

N Engl J Med. 2007 Mar 15;356(11):1094-7. No abstract available.

PMID:
17360987
17.

Combinatorial effects of multiple enhancer variants in linkage disequilibrium dictate levels of gene expression to confer susceptibility to common traits.

Corradin O, Saiakhova A, Akhtar-Zaidi B, Myeroff L, Willis J, Cowper-Sal lari R, Lupien M, Markowitz S, Scacheri PC.

Genome Res. 2014 Jan;24(1):1-13. doi: 10.1101/gr.164079.113. Epub 2013 Nov 6.

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No association between common variants in glyoxalase 1 and autism spectrum disorders.

Rehnström K, Ylisaukko-Oja T, Vanhala R, von Wendt L, Peltonen L, Hovatta I.

Am J Med Genet B Neuropsychiatr Genet. 2008 Jan 5;147B(1):124-7.

PMID:
17722011
20.

Variants associated with common disease are not unusually differentiated in frequency across populations.

Lohmueller KE, Mauney MM, Reich D, Braverman JM.

Am J Hum Genet. 2006 Jan;78(1):130-6. Epub 2005 Nov 16.

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