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p19ARF-independent induction of p53 and cell cycle arrest by Raf in murine keratinocytes.

Roper E, Weinberg W, Watt FM, Land H.

EMBO Rep. 2001 Feb;2(2):145-50.


Defects in TGF-beta signaling overcome senescence of mouse keratinocytes expressing v-Ha-ras.

Tremain R, Marko M, Kinnimulki V, Ueno H, Bottinger E, Glick A.

Oncogene. 2000 Mar 23;19(13):1698-709.


Myc antagonizes Ras-mediated growth arrest in leukemia cells through the inhibition of the Ras-ERK-p21Cip1 pathway.

Vaqué JP, Navascues J, Shiio Y, Laiho M, Ajenjo N, Mauleon I, Matallanas D, Crespo P, León J.

J Biol Chem. 2005 Jan 14;280(2):1112-22. Epub 2004 Nov 4.


H-, K- and N-Ras inhibit myeloid leukemia cell proliferation by a p21WAF1-dependent mechanism.

Delgado MD, Vaqué JP, Arozarena I, López-Ilasaca MA, Martínez C, Crespo P, León J.

Oncogene. 2000 Feb 10;19(6):783-90.


Modulation of p53, ErbB1, ErbB2, and Raf-1 expression in lung cancer cells by depsipeptide FR901228.

Yu X, Guo ZS, Marcu MG, Neckers L, Nguyen DM, Chen GA, Schrump DS.

J Natl Cancer Inst. 2002 Apr 3;94(7):504-13.


Efficient down-regulation of cyclin A-associated activity and expression in suspended primary keratinocytes requires p21(Cip1).

Hauser P, Ma L, Agrawal D, Haura E, Cress WD, Pledger WJ.

Mol Cancer Res. 2004 Feb;2(2):96-104.


Dual growth arrest pathways in astrocytes and astrocytic tumors in response to Raf-1 activation.

Fanton CP, McMahon M, Pieper RO.

J Biol Chem. 2001 Jun 1;276(22):18871-7. Epub 2001 Mar 15.


High-intensity Raf signal causes cell cycle arrest mediated by p21Cip1.

Sewing A, Wiseman B, Lloyd AC, Land H.

Mol Cell Biol. 1997 Sep;17(9):5588-97.


Mechanisms of Taxol-induced cell death are concentration dependent.

Torres K, Horwitz SB.

Cancer Res. 1998 Aug 15;58(16):3620-6.


p19ARF directly and differentially controls the functions of c-Myc independently of p53.

Qi Y, Gregory MA, Li Z, Brousal JP, West K, Hann SR.

Nature. 2004 Oct 7;431(7009):712-7. Epub 2004 Sep 8.


Inhibitory function of p21Cip1/WAF1 in differentiation of primary mouse keratinocytes independent of cell cycle control.

Di Cunto F, Topley G, Calautti E, Hsiao J, Ong L, Seth PK, Dotto GP.

Science. 1998 May 15;280(5366):1069-72.


Taxol induction of p21WAF1 and p53 requires c-raf-1.

Blagosklonny MV, Schulte TW, Nguyen P, Mimnaugh EG, Trepel J, Neckers L.

Cancer Res. 1995 Oct 15;55(20):4623-6.


A transcriptional activation function of p53 is dispensable for and inhibitory of its apoptotic function.

Kokontis JM, Wagner AJ, O'Leary M, Liao S, Hay N.

Oncogene. 2001 Feb 8;20(6):659-68.


Oridonin induces G2/M arrest and apoptosis via activating ERK-p53 apoptotic pathway and inhibiting PTK-Ras-Raf-JNK survival pathway in murine fibrosarcoma L929 cells.

Cheng Y, Qiu F, Ye YC, Tashiro S, Onodera S, Ikejima T.

Arch Biochem Biophys. 2009 Oct 1;490(1):70-5. doi: 10.1016/ Epub 2009 Aug 20.


Inhibition of v-Abl transformation by p53 and p19ARF.

Cong F, Zou X, Hinrichs K, Calame K, Goff SP.

Oncogene. 1999 Dec 16;18(54):7731-9.


Endogenous p53 gene status predicts the response of human squamous cell carcinomas to wild-type p53.

St John LS, Sauter ER, Herlyn M, Litwin S, Adler-Storthz K.

Cancer Gene Ther. 2000 May;7(5):749-56.


The absence of p21Cip1/WAF1 alters keratinocyte growth and differentiation and promotes ras-tumor progression.

Missero C, Di Cunto F, Kiyokawa H, Koff A, Dotto GP.

Genes Dev. 1996 Dec 1;10(23):3065-75.


Cyclosporin A-induced hair growth in mice is associated with inhibition of calcineurin-dependent activation of NFAT in follicular keratinocytes.

Gafter-Gvili A, Sredni B, Gal R, Gafter U, Kalechman Y.

Am J Physiol Cell Physiol. 2003 Jun;284(6):C1593-603.


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