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Items: 1 to 20 of 102

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Elimination of replication block protein Fob1 extends the life span of yeast mother cells.

Defossez PA, Prusty R, Kaeberlein M, Lin SJ, Ferrigno P, Silver PA, Keil RL, Guarente L.

Mol Cell. 1999 Apr;3(4):447-55.

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The RAD52 epistasis group in mammalian double strand break repair.

Petrini JH, Bressan DA, Yao MS.

Semin Immunol. 1997 Jun;9(3):181-8. Review.

PMID:
9200329
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Silencing factors participate in DNA repair and recombination in Saccharomyces cerevisiae.

Tsukamoto Y, Kato J, Ikeda H.

Nature. 1997 Aug 28;388(6645):900-3.

PMID:
9278054
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The Escherichia coli RecA protein complements recombination defective phenotype of the Saccharomyces cerevisiae rad52 mutant cells.

Dudás A, Marková E, Vlasáková D, Kolman A, Bartosová Z, Brozmanová J, Chovanec M.

Yeast. 2003 Apr 15;20(5):389-96.

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Ionizing radiation-induced foci formation of mammalian Rad51 and Rad54 depends on the Rad51 paralogs, but not on Rad52.

van Veelen LR, Essers J, van de Rakt MW, Odijk H, Pastink A, Zdzienicka MZ, Paulusma CC, Kanaar R.

Mutat Res. 2005 Jul 1;574(1-2):34-49. Epub 2005 Apr 9.

PMID:
15914205
18.

Role of RAD52 epistasis group genes in homologous recombination and double-strand break repair.

Symington LS.

Microbiol Mol Biol Rev. 2002 Dec;66(4):630-70, table of contents. Review.

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Rad52 forms DNA repair and recombination centers during S phase.

Lisby M, Rothstein R, Mortensen UH.

Proc Natl Acad Sci U S A. 2001 Jul 17;98(15):8276-82.

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Molecular dissection of interactions between Rad51 and members of the recombination-repair group.

Krejci L, Damborsky J, Thomsen B, Duno M, Bendixen C.

Mol Cell Biol. 2001 Feb;21(3):966-76.

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