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Items: 1 to 20 of 82

1.

Chk2 is required for HSV-1 ICP0-mediated G2/M arrest and enhancement of virus growth.

Li H, Baskaran R, Krisky DM, Bein K, Grandi P, Cohen JB, Glorioso JC.

Virology. 2008 May 25;375(1):13-23. doi: 10.1016/j.virol.2008.01.038. Epub 2008 Mar 5.

2.

Cellular Transcriptional Coactivator RanBP10 and Herpes Simplex Virus 1 ICP0 Interact and Synergistically Promote Viral Gene Expression and Replication.

Sato Y, Kato A, Maruzuru Y, Oyama M, Kozuka-Hata H, Arii J, Kawaguchi Y.

J Virol. 2016 Jan 6;90(6):3173-86. doi: 10.1128/JVI.03043-15.

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A pre-immediate-early role for tegument ICP0 in the proteasome-dependent entry of herpes simplex virus.

Delboy MG, Nicola AV.

J Virol. 2011 Jun;85(12):5910-8. doi: 10.1128/JVI.00267-11. Epub 2011 Apr 6.

13.

Both ERK1 and ERK2 kinases promote G2/M arrest in etoposide-treated MCF7 cells by facilitating ATM activation.

Wei F, Xie Y, Tao L, Tang D.

Cell Signal. 2010 Nov;22(11):1783-9. doi: 10.1016/j.cellsig.2010.07.007. Epub 2010 Jul 15.

PMID:
20637859
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Sulforaphane-induced G2/M phase cell cycle arrest involves checkpoint kinase 2-mediated phosphorylation of cell division cycle 25C.

Singh SV, Herman-Antosiewicz A, Singh AV, Lew KL, Srivastava SK, Kamath R, Brown KD, Zhang L, Baskaran R.

J Biol Chem. 2004 Jun 11;279(24):25813-22. Epub 2004 Apr 8.

17.

ICP0 and the US3 protein kinase of herpes simplex virus 1 independently block histone deacetylation to enable gene expression.

Poon AP, Gu H, Roizman B.

Proc Natl Acad Sci U S A. 2006 Jun 27;103(26):9993-8. Epub 2006 Jun 19.

18.

The ATM/ATR signaling effector Chk2 is targeted by Epstein-Barr virus nuclear antigen 3C to release the G2/M cell cycle block.

Choudhuri T, Verma SC, Lan K, Murakami M, Robertson ES.

J Virol. 2007 Jun;81(12):6718-30. Epub 2007 Apr 4.

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The stability of herpes simplex virus 1 ICP0 early after infection is defined by the RING finger and the UL13 protein kinase.

Zhu Z, Du T, Zhou G, Roizman B.

J Virol. 2014 May;88(10):5437-43. doi: 10.1128/JVI.00542-14. Epub 2014 Feb 26.

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