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Items: 7

1.

High-throughput gene expression analysis identifies p53-dependent and -independent pathways contributing to the adrenocortical dysplasia (acd) phenotype.

Sucularli C, Thomas P, Kocak H, White JS, O'Connor BC, Keegan CE.

Gene. 2018 Dec 30;679:219-231. doi: 10.1016/j.gene.2018.09.002. Epub 2018 Sep 4.

PMID:
30189268
2.

Silencing of telomere-binding protein adrenocortical dysplasia (ACD) homolog enhances radiosensitivity in glioblastoma cells.

Polito F, Cucinotta M, Abbritti RV, Brogna A, Pergolizzi S, Tomasello C, Barresi V, Angileri FF, Di Giorgio R, Conti A, La Torre D, Germanò A, Aguennouz M.

Transl Res. 2018 Dec;202:99-108. doi: 10.1016/j.trsl.2018.07.005. Epub 2018 Jul 20.

PMID:
30080989
3.

Use of a GnRH vaccine, GonaCon, for prevention and treatment of adrenocortical disease (ACD) in domestic ferrets.

Miller LA, Fagerstone KA, Wagner RA, Finkler M.

Vaccine. 2013 Sep 23;31(41):4619-23. doi: 10.1016/j.vaccine.2013.07.035. Epub 2013 Jul 29.

PMID:
23906891
4.

Additive effect of TAp63 deficiency on the adrenocortical dysplasia (acd) phenotype.

O'Connor BC, Macke EL, Keegan CE.

Mamm Genome. 2011 Dec;22(11-12):714-21. doi: 10.1007/s00335-011-9360-4. Epub 2011 Oct 22.

5.

Caudal regression in adrenocortical dysplasia (acd) mice is caused by telomere dysfunction with subsequent p53-dependent apoptosis.

Vlangos CN, O'Connor BC, Morley MJ, Krause AS, Osawa GA, Keegan CE.

Dev Biol. 2009 Oct 15;334(2):418-28. doi: 10.1016/j.ydbio.2009.07.038. Epub 2009 Aug 3.

6.

Genetic p53 deficiency partially rescues the adrenocortical dysplasia phenotype at the expense of increased tumorigenesis.

Else T, Trovato A, Kim AC, Wu Y, Ferguson DO, Kuick RD, Lucas PC, Hammer GD.

Cancer Cell. 2009 Jun 2;15(6):465-76. doi: 10.1016/j.ccr.2009.04.011.

7.

Clinical and endocrine responses to treatment with deslorelin acetate implants in ferrets with adrenocortical disease.

Wagner RA, Piché CA, Jöchle W, Oliver JW.

Am J Vet Res. 2005 May;66(5):910-4.

PMID:
15934621

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