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1965 1
1969 4
1970 3
1971 4
1972 3
1973 2
1974 4
1975 23
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1977 31
1978 33
1979 38
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1981 38
1982 50
1983 54
1984 59
1985 56
1986 62
1987 65
1988 62
1989 100
1990 95
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1992 126
1993 113
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1995 119
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1998 135
1999 136
2000 169
2001 177
2002 168
2003 150
2004 194
2005 230
2006 223
2007 261
2008 272
2009 233
2010 268
2011 213
2012 249
2013 301
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The following term was not found in PubMed: 003590475.1
Page 1
Xeroderma Pigmentosum.
Kraemer KH, DiGiovanna JJ, Tamura D. Kraemer KH, et al. 2003 Jun 20 [updated 2022 Mar 24]. In: Adam MP, Feldman J, Mirzaa GM, Pagon RA, Wallace SE, Bean LJH, Gripp KW, Amemiya A, editors. GeneReviews® [Internet]. Seattle (WA): University of Washington, Seattle; 1993–2024. 2003 Jun 20 [updated 2022 Mar 24]. In: Adam MP, Feldman J, Mirzaa GM, Pagon RA, Wallace SE, Bean LJH, Gripp KW, Amemiya A, editors. GeneReviews® [Internet]. Seattle (WA): University of Washington, Seattle; 1993–2024. PMID: 20301571 Free Books & Documents. Review.
The most common causes of death are skin cancer, neurologic degeneration, and internal cancer. The median age at death in persons with XP with neurodegeneration (29 years) was found to be younger than that in persons with XP without neurodegeneration (37 years). ... …
The most common causes of death are skin cancer, neurologic degeneration, and internal cancer. The median age at death in persons with XP
Xylan Plastic.
Jia S, Lv Z, Rao J, Lü B, Chen G, Bian J, Li M, Peng F. Jia S, et al. ACS Nano. 2023 Jul 25;17(14):13627-13637. doi: 10.1021/acsnano.3c02327. Epub 2023 Jul 13. ACS Nano. 2023. PMID: 37439501
Moreover, XP showed a low thermal expansion coefficient and excellent optical properties. Cytotoxicity and degradability tests demonstrated that XP had excellent nontoxicity and can be biodegraded in 60 days. ...
Moreover, XP showed a low thermal expansion coefficient and excellent optical properties. Cytotoxicity and degradability tests demons …
Xeroderma pigmentosum-Cockayne syndrome complex.
Natale V, Raquer H. Natale V, et al. Orphanet J Rare Dis. 2017 Apr 4;12(1):65. doi: 10.1186/s13023-017-0616-2. Orphanet J Rare Dis. 2017. PMID: 28376890 Free PMC article. Review.
There are four groups in XP-CS, and classification was available for 42 patients. Twenty-one were in the XP-G complementation group, 13 in XP-D, 5 in XP-B, and 3 in XP-F. ...However, one intriguing finding was that cancer incidence was lower in …
There are four groups in XP-CS, and classification was available for 42 patients. Twenty-one were in the XP-G complementation …
Sunlight, Vitamin D, and Xeroderma Pigmentosum.
Martens MC, Emmert S, Boeckmann L. Martens MC, et al. Adv Exp Med Biol. 2020;1268:319-331. doi: 10.1007/978-3-030-46227-7_16. Adv Exp Med Biol. 2020. PMID: 32918226 Review.
As UV radiation plays a critical role in the photosynthesis of vitamin D, stringent sun protection, as recommended for xeroderma pigmentosum (XP) patients, may impact their vitamin D levels.XP is a rare autosomal recessive disorder with a worldwide prevalence of 1 i …
As UV radiation plays a critical role in the photosynthesis of vitamin D, stringent sun protection, as recommended for xeroderma pigmentosum …
Simulational Tests of the Rouse Model.
Phillies GDJ. Phillies GDJ. Polymers (Basel). 2023 Jun 8;15(12):2615. doi: 10.3390/polym15122615. Polymers (Basel). 2023. PMID: 37376261 Free PMC article. Review.
We focus on Rouse model predictions for the mean-square amplitudes (Xp(0))2 and time correlation functions Xp(0)Xp(t) of the Rouse mode Xp(t). ...For small p (say, p3) (Xp(0))2 scales with p as p-2; for larger p, it scales as p-3. (ii) Rouse mod …
We focus on Rouse model predictions for the mean-square amplitudes (Xp(0))2 and time correlation functions Xp(0)Xp(t) o …
Xeroderma Pigmentosum: A Model for Human Premature Aging.
Rizza ERH, DiGiovanna JJ, Khan SG, Tamura D, Jeskey JD, Kraemer KH. Rizza ERH, et al. J Invest Dermatol. 2021 Apr;141(4S):976-984. doi: 10.1016/j.jid.2020.11.012. Epub 2021 Jan 9. J Invest Dermatol. 2021. PMID: 33436302 Free PMC article. Review.
Poikiloderma, atypical lentigines, and skin cancers, the primary cutaneous features of XP, occur in the general population but at a much older age. Patients with XP also exhibit ocular changes secondary to premature photoaging, including ocular surface tumors and pt …
Poikiloderma, atypical lentigines, and skin cancers, the primary cutaneous features of XP, occur in the general population but at a m …
Meta-analysis of three randomized trials of capecitabine plus cisplatin (XP) versus S-1 plus cisplatin (SP) as first-line treatment for advanced gastric cancer.
Nishikawa K, Kawakami H, Shimokawa T, Fujitani K, Tamura S, Endo S, Kobayashi M, Kawada J, Kurokawa Y, Tsuburaya A, Yoshikawa T, Sakamoto J, Satoh T; for HERBIS-2, HERBIS-4A, XParTS I. I. study investigators. Nishikawa K, et al. Int J Clin Oncol. 2023 Nov;28(11):1501-1510. doi: 10.1007/s10147-023-02402-1. Epub 2023 Aug 27. Int J Clin Oncol. 2023. PMID: 37634209
BACKGROUND: S-1 plus cisplatin (SP) and capecitabine plus cisplatin (XP) are standard first-line regimens for advanced gastric cancer (AGC) worldwide. ...For the differentiated type, OS was significantly longer in the SP group (13.2 months) than in the XP group (11. …
BACKGROUND: S-1 plus cisplatin (SP) and capecitabine plus cisplatin (XP) are standard first-line regimens for advanced gastric cancer …
Shining a light on xeroderma pigmentosum.
DiGiovanna JJ, Kraemer KH. DiGiovanna JJ, et al. J Invest Dermatol. 2012 Mar;132(3 Pt 2):785-96. doi: 10.1038/jid.2011.426. Epub 2012 Jan 5. J Invest Dermatol. 2012. PMID: 22217736 Free PMC article. Review.
XP cells were found to have defects in seven of the proteins of the nucleotide excision repair pathway and in DNA polymerase eta. XP cells are hypersensitive to killing by UV radiation, and XP cancers have characteristic "UV signature" mutations. ...
XP cells were found to have defects in seven of the proteins of the nucleotide excision repair pathway and in DNA polymerase eta.
Nucleotide Excision Repair: From Molecular Defects to Neurological Abnormalities.
Krasikova Y, Rechkunova N, Lavrik O. Krasikova Y, et al. Int J Mol Sci. 2021 Jun 9;22(12):6220. doi: 10.3390/ijms22126220. Int J Mol Sci. 2021. PMID: 34207557 Free PMC article. Review.
A specific class of oxidatively induced DNA lesions, 8,5'-cyclopurine-2'-deoxynucleosides, is considered endogenous DNA lesions mainly responsible for neurological problems in XP. Growing evidence suggests that XP is accompanied by defective mitophagy, as in primary …
A specific class of oxidatively induced DNA lesions, 8,5'-cyclopurine-2'-deoxynucleosides, is considered endogenous DNA lesions mainly respo …
Xeroderma pigmentosum and skin cancer.
Daya-Grosjean L. Daya-Grosjean L. Adv Exp Med Biol. 2008;637:19-27. doi: 10.1007/978-0-387-09599-8_3. Adv Exp Med Biol. 2008. PMID: 19181107 Review.
Indeed, XP presents a unique model for analysing the effects of unrepaired DNA lesions in skin carcinogenesis. The skin cancer predisposition, observed in XP patients, is due to the mutator gene activity of XP cells which lead to high levels of UV specific mo …
Indeed, XP presents a unique model for analysing the effects of unrepaired DNA lesions in skin carcinogenesis. The skin cancer predis …
8,429 results