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Items: 1 to 20 of 56

1.

Biological Activities, Health Benefits, and Therapeutic Properties of Avenanthramides: From Skin Protection to Prevention and Treatment of Cerebrovascular Diseases.

Perrelli A, Goitre L, Salzano AM, Moglia A, Scaloni A, Retta SF.

Oxid Med Cell Longev. 2018 Aug 23;2018:6015351. doi: 10.1155/2018/6015351. eCollection 2018. Review.

2.

Yeast-Derived Recombinant Avenanthramides Inhibit Proliferation, Migration and Epithelial Mesenchymal Transition of Colon Cancer Cells.

Finetti F, Moglia A, Schiavo I, Donnini S, Berta GN, Di Scipio F, Perrelli A, Fornelli C, Trabalzini L, Retta SF.

Nutrients. 2018 Aug 24;10(9). pii: E1159. doi: 10.3390/nu10091159.

3.

Data in support of sustained upregulation of adaptive redox homeostasis mechanisms caused by KRIT1 loss-of-function.

Antognelli C, Trapani E, Delle Monache S, Perrelli A, Fornelli C, Retta F, Cassoni P, Talesa VN, Retta SF.

Data Brief. 2017 Dec 13;16:929-938. doi: 10.1016/j.dib.2017.12.026. eCollection 2018 Feb.

4.

KRIT1 loss-of-function induces a chronic Nrf2-mediated adaptive homeostasis that sensitizes cells to oxidative stress: Implication for Cerebral Cavernous Malformation disease.

Antognelli C, Trapani E, Delle Monache S, Perrelli A, Daga M, Pizzimenti S, Barrera G, Cassoni P, Angelucci A, Trabalzini L, Talesa VN, Goitre L, Retta SF.

Free Radic Biol Med. 2018 Feb 1;115:202-218. doi: 10.1016/j.freeradbiomed.2017.11.014. Epub 2017 Nov 21.

5.

Up-regulation of NADPH oxidase-mediated redox signaling contributes to the loss of barrier function in KRIT1 deficient endothelium.

Goitre L, DiStefano PV, Moglia A, Nobiletti N, Baldini E, Trabalzini L, Keubel J, Trapani E, Shuvaev VV, Muzykantov VR, Sarelius IH, Retta SF, Glading AJ.

Sci Rep. 2017 Aug 15;7(1):8296. doi: 10.1038/s41598-017-08373-4.

6.

Oxidative stress and inflammation in cerebral cavernous malformation disease pathogenesis: Two sides of the same coin.

Retta SF, Glading AJ.

Int J Biochem Cell Biol. 2016 Dec;81(Pt B):254-270. doi: 10.1016/j.biocel.2016.09.011. Epub 2016 Sep 14. Review.

7.

Beyond multiple mechanisms and a unique drug: Defective autophagy as pivotal player in cerebral cavernous malformation pathogenesis and implications for targeted therapies.

Marchi S, Trapani E, Corricelli M, Goitre L, Pinton P, Retta SF.

Rare Dis. 2016 Jan 25;4(1):e1142640. doi: 10.1080/21675511.2016.1142640. eCollection 2016.

8.

Cellular processes underlying cerebral cavernous malformations: Autophagy as another point of view.

Marchi S, Retta SF, Pinton P.

Autophagy. 2016;12(2):424-5. doi: 10.1080/15548627.2015.1125073.

9.

Platinum nanozymes recover cellular ROS homeostasis in an oxidative stress-mediated disease model.

Moglianetti M, De Luca E, Pedone D, Marotta R, Catelani T, Sartori B, Amenitsch H, Retta SF, Pompa PP.

Nanoscale. 2016 Feb 14;8(6):3739-52. doi: 10.1039/c5nr08358c. Epub 2016 Jan 27.

PMID:
26815950
10.

Cytochrome P450 and matrix metalloproteinase genetic modifiers of disease severity in Cerebral Cavernous Malformation type 1.

Choquet H, Trapani E, Goitre L, Trabalzini L, Akers A, Fontanella M, Hart BL, Morrison LA, Pawlikowska L, Kim H, Retta SF.

Free Radic Biol Med. 2016 Mar;92:100-109. doi: 10.1016/j.freeradbiomed.2016.01.008. Epub 2016 Jan 19.

11.

Defective autophagy is a key feature of cerebral cavernous malformations.

Marchi S, Corricelli M, Trapani E, Bravi L, Pittaro A, Delle Monache S, Ferroni L, Patergnani S, Missiroli S, Goitre L, Trabalzini L, Rimessi A, Giorgi C, Zavan B, Cassoni P, Dejana E, Retta SF, Pinton P.

EMBO Mol Med. 2015 Nov;7(11):1403-17. doi: 10.15252/emmm.201505316.

12.

Cerebral cavernous malformation (CCM) disease: from monogenic forms to genetic susceptibility factors.

Trapani E, Retta SF.

J Neurosurg Sci. 2015 Sep;59(3):201-9. Epub 2015 Apr 21. Review.

PMID:
25896717
13.

Yersinia enterocolitica exploits different pathways to accomplish adhesion and toxin injection into host cells.

Keller B, Mühlenkamp M, Deuschle E, Siegfried A, Mössner S, Schade J, Griesinger T, Katava N, Braunsdorf C, Fehrenbacher B, Jiménez-Soto LF, Schaller M, Haas R, Genth H, Retta SF, Meyer H, Böttcher RT, Zent R, Schütz M, Autenrieth IB, Bohn E.

Cell Microbiol. 2015 Aug;17(8):1179-204. doi: 10.1111/cmi.12429. Epub 2015 Mar 20.

PMID:
25678064
14.

Evaluation of the bioactive properties of avenanthramide analogs produced in recombinant yeast.

Moglia A, Goitre L, Gianoglio S, Baldini E, Trapani E, Genre A, Scattina A, Dondo G, Trabalzini L, Beekwilder J, Retta SF.

Biofactors. 2015 Jan-Feb;41(1):15-27. doi: 10.1002/biof.1197. Epub 2015 Jan 30.

15.

Strategy for identifying repurposed drugs for the treatment of cerebral cavernous malformation.

Gibson CC, Zhu W, Davis CT, Bowman-Kirigin JA, Chan AC, Ling J, Walker AE, Goitre L, Delle Monache S, Retta SF, Shiu YT, Grossmann AH, Thomas KR, Donato AJ, Lesniewski LA, Whitehead KJ, Li DY.

Circulation. 2015 Jan 20;131(3):289-99. doi: 10.1161/CIRCULATIONAHA.114.010403. Epub 2014 Dec 8.

16.

Fluorescence microscopy study of Rap1 subcellular localization.

Goitre L, Cutano V, Retta SF.

Methods Mol Biol. 2014;1120:197-205. doi: 10.1007/978-1-62703-791-4_13.

PMID:
24470027
17.

Combined pulldown and time-lapse microscopy studies for determining the role of Rap1 in the crosstalk between integrins and cadherins.

Goitre L, Retta SF.

Methods Mol Biol. 2014;1120:177-95. doi: 10.1007/978-1-62703-791-4_12.

PMID:
24470026
18.

Ras GTPases are both regulators and effectors of redox agents.

Ferro E, Goitre L, Baldini E, Retta SF, Trabalzini L.

Methods Mol Biol. 2014;1120:55-74. doi: 10.1007/978-1-62703-791-4_5. Review.

PMID:
24470019
19.

The Ras superfamily of small GTPases: the unlocked secrets.

Goitre L, Trapani E, Trabalzini L, Retta SF.

Methods Mol Biol. 2014;1120:1-18. doi: 10.1007/978-1-62703-791-4_1. Review.

PMID:
24470015
20.

KRIT1 loss of function causes a ROS-dependent upregulation of c-Jun.

Goitre L, De Luca E, Braggion S, Trapani E, Guglielmotto M, Biasi F, Forni M, Moglia A, Trabalzini L, Retta SF.

Free Radic Biol Med. 2014 Mar;68:134-47. doi: 10.1016/j.freeradbiomed.2013.11.020. Epub 2013 Nov 28.

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