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Page 1
The mechanism underlying transient weakness in myotonia congenita.
Elife. 2021 Apr 27;10:e65691. doi: 10.7554/eLife.65691.
Elife. 2021.
PMID: 33904400
Free PMC article.
Open-label trial of ranolazine for the treatment of myotonia congenita.
Arnold WD, Kline D, Sanderson A, Hawash AA, Bartlett A, Novak KR, Rich MM, Kissel JT.
Arnold WD, et al. Among authors: novak kr.
Neurology. 2017 Aug 15;89(7):710-713. doi: 10.1212/WNL.0000000000004229. Epub 2017 Jul 14.
Neurology. 2017.
PMID: 28710329
Free PMC article.
Clinical Trial.
Item in Clipboard
Altered sodium channel-protein associations in critical illness myopathy.
Kraner SD, Novak KR, Wang Q, Peng J, Rich MM.
Kraner SD, et al. Among authors: novak kr.
Skelet Muscle. 2012 Aug 30;2(1):17. doi: 10.1186/2044-5040-2-17.
Skelet Muscle. 2012.
PMID: 22935229
Free PMC article.
Item in Clipboard
Sodium channel slow inactivation as a therapeutic target for myotonia congenita.
Novak KR, Norman J, Mitchell JR, Pinter MJ, Rich MM.
Novak KR, et al.
Ann Neurol. 2015 Feb;77(2):320-32. doi: 10.1002/ana.24331. Epub 2015 Jan 9.
Ann Neurol. 2015.
PMID: 25515836
Free PMC article.
Item in Clipboard
Upregulation of the CaV 1.1-ryanodine receptor complex in a rat model of critical illness myopathy.
Kraner SD, Wang Q, Novak KR, Cheng D, Cool DR, Peng J, Rich MM.
Kraner SD, et al. Among authors: novak kr.
Am J Physiol Regul Integr Comp Physiol. 2011 Jun;300(6):R1384-91. doi: 10.1152/ajpregu.00032.2011. Epub 2011 Apr 6.
Am J Physiol Regul Integr Comp Physiol. 2011.
PMID: 21474431
Free PMC article.
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Inactivation of sodium channels underlies reversible neuropathy during critical illness in rats.
Novak KR, Nardelli P, Cope TC, Filatov G, Glass JD, Khan J, Rich MM.
Novak KR, et al.
J Clin Invest. 2009 May;119(5):1150-8. doi: 10.1172/jci36570.
J Clin Invest. 2009.
PMID: 19425168
Free PMC article.
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