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Page 1
FSHD1 and FSHD2 form a disease continuum.
Neurology. 2019 May 7;92(19):e2273-e2285. doi: 10.1212/WNL.0000000000007456. Epub 2019 Apr 12.
Neurology. 2019.
PMID: 30979860
Free PMC article.
The Landscape of L1 Retrotransposons in the Human Genome Is Shaped by Pre-insertion Sequence Biases and Post-insertion Selection.
Sultana T, van Essen D, Siol O, Bailly-Bechet M, Philippe C, Zine El Aabidine A, Pioger L, Nigumann P, Saccani S, Andrau JC, Gilbert N, Cristofari G.
Sultana T, et al. Among authors: nigumann p.
Mol Cell. 2019 May 2;74(3):555-570.e7. doi: 10.1016/j.molcel.2019.02.036. Epub 2019 Apr 4.
Mol Cell. 2019.
PMID: 30956044
Free article.
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Activation of individual L1 retrotransposon instances is restricted to cell-type dependent permissive loci.
Philippe C, Vargas-Landin DB, Doucet AJ, van Essen D, Vera-Otarola J, Kuciak M, Corbin A, Nigumann P, Cristofari G.
Philippe C, et al. Among authors: nigumann p.
Elife. 2016 Mar 26;5:e13926. doi: 10.7554/eLife.13926.
Elife. 2016.
PMID: 27016617
Free PMC article.
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Many human genes are transcribed from the antisense promoter of L1 retrotransposon.
Nigumann P, Redik K, Mätlik K, Speek M.
Nigumann P, et al.
Genomics. 2002 May;79(5):628-34. doi: 10.1006/geno.2002.6758.
Genomics. 2002.
PMID: 11991712
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Intronic L1 retrotransposons and nested genes cause transcriptional interference by inducing intron retention, exonization and cryptic polyadenylation.
Kaer K, Branovets J, Hallikma A, Nigumann P, Speek M.
Kaer K, et al. Among authors: nigumann p.
PLoS One. 2011;6(10):e26099. doi: 10.1371/journal.pone.0026099. Epub 2011 Oct 13.
PLoS One. 2011.
PMID: 22022525
Free PMC article.
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