Format

Send to

Choose Destination

See 1 citation found by title matching your search:

PM R. 2015 Jul;7(7):746-61. doi: 10.1016/j.pmrj.2015.01.024. Epub 2015 Feb 24.

Myofascial Trigger Points Then and Now: A Historical and Scientific Perspective.

Author information

1
Rehabilitation Medicine Department, Clinical Center, National Institutes of Health, 9000 Rockville Pike, Bethesda, MD 20892(∗). Electronic address: jshah@mail.cc.nih.gov.
2
Rehabilitation Medicine Department, Clinical Center, National Institutes of Health, Bethesda, MD(†).
3
Rehabilitation Medicine Department, Clinical Center, National Institutes of Health, Bethesda, MD(‡).
4
Rehabilitation Medicine Department, Clinical Center, National Institutes of Health, Bethesda, MD(§).
5
Departments of Electrical and Computer Engineering and Bioengineering, Volgenau School of Engineering, George Mason University, Fairfax, VA(‖).
6
Center for the Study of Chronic Illness and Disability, College of Health and Human Services, George Mason University, Fairfax, VA(¶).

Abstract

The intent of this article is to discuss the evolving role of the myofascial trigger point (MTrP) in myofascial pain syndrome (MPS) from both a historical and scientific perspective. MTrPs are hard, discrete, palpable nodules in a taut band of skeletal muscle that may be spontaneously painful (i.e., active) or painful only on compression (i.e., latent). MPS is a term used to describe a pain condition that can be acute or, more commonly, chronic and involves the muscle and its surrounding connective tissue (e.g. fascia). According to Travell and Simons, MTrPs are central to the syndrome-but are they necessary? Although the clinical study of muscle pain and MTrPs has proliferated over the past two centuries, the scientific literature often seems disjointed and confusing. Unfortunately, much of the terminology, theories, concepts, and diagnostic criteria are inconsistent, incomplete, or controversial. To address these deficiencies, investigators have recently applied clinical, imaging (of skeletal muscle and brain), and biochemical analyses to systematically and objectively study the MTrP and its role in MPS. Data suggest that the soft tissue milieu around the MTrP, neurogenic inflammation, sensitization, and limbic system dysfunction may all play a role in the initiation, amplification, and perpetuation of MPS. The authors chronicle the advances that have led to the current understanding of MTrP pathophysiology and its relationship to MPS, and review the contributions of clinicians and researchers who have influenced and expanded our contemporary level of clinical knowledge and practice.

PMID:
25724849
PMCID:
PMC4508225
DOI:
10.1016/j.pmrj.2015.01.024
[Indexed for MEDLINE]
Free PMC Article

Supplemental Content

Full text links

Icon for Elsevier Science Icon for PubMed Central
Loading ...
Support Center