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Items: 13

1.

Pregnancy-adapted uterine artery endothelial cell Ca2+ signaling and its relationship with membrane potential.

Alvarez RE, Boeldt DS, Pattnaik BR, Friedman HL, Bird IM.

Physiol Rep. 2017 Nov;5(21). pii: e13452. doi: 10.14814/phy2.13452.

2.

Positive versus negative effects of VEGF165 on Ca2+ signaling and NO production in human endothelial cells.

Boeldt DS, Krupp J, Yi FX, Khurshid N, Shah DM, Bird IM.

Am J Physiol Heart Circ Physiol. 2017 Jan 1;312(1):H173-H181. doi: 10.1152/ajpheart.00924.2015. Epub 2016 Nov 11.

3.

Vascular adaptation in pregnancy and endothelial dysfunction in preeclampsia.

Boeldt DS, Bird IM.

J Endocrinol. 2017 Jan;232(1):R27-R44. Epub 2016 Oct 11. Review.

4.

Changes in Ca2+ Signaling and Nitric Oxide Output by Human Umbilical Vein Endothelium in Diabetic and Gestational Diabetic Pregnancies.

Anaya HA, Yi FX, Boeldt DS, Krupp J, Grummer MA, Shah DM, Bird IM.

Biol Reprod. 2015 Sep;93(3):60. doi: 10.1095/biolreprod.115.128645. Epub 2015 Jul 22.

5.

Phosphorylation of Ser-279/282 and Tyr-265 positions on Cx43 as possible mediators of VEGF-165 inhibition of pregnancy-adapted Ca2+ burst function in ovine uterine artery endothelial cells.

Boeldt DS, Grummer MA, Yi F, Magness RR, Bird IM.

Mol Cell Endocrinol. 2015 Sep 5;412:73-84. doi: 10.1016/j.mce.2015.05.030. Epub 2015 May 29.

6.

Altered VEGF-stimulated Ca2+ signaling in part underlies pregnancy-adapted eNOS activity in UAEC.

Boeldt DS, Grummer MA, Magness RR, Bird IM.

J Endocrinol. 2014 Oct;223(1):1-11. doi: 10.1530/JOE-14-0252. Epub 2014 Jul 25.

7.

Pregnancy programming and preeclampsia: identifying a human endothelial model to study pregnancy-adapted endothelial function and endothelial adaptive failure in preeclamptic subjects.

Boeldt DS, Hankes AC, Alvarez RE, Khurshid N, Balistreri M, Grummer MA, Yi F, Bird IM.

Adv Exp Med Biol. 2014;814:27-47. doi: 10.1007/978-1-4939-1031-1_4. Review.

PMID:
25015799
8.

Pregnancy, programming and preeclampsia: gap junctions at the nexus of pregnancy-induced adaptation of endothelial function and endothelial adaptive failure in PE.

Bird IM, Boeldt DS, Krupp J, Grummer MA, Yi FX, Magness RR.

Curr Vasc Pharmacol. 2013 Sep;11(5):712-29. Review.

PMID:
24063383
9.

The loss of sustained Ca(2+) signaling underlies suppressed endothelial nitric oxide production in preeclamptic pregnancies: implications for new therapy.

Krupp J, Boeldt DS, Yi FX, Grummer MA, Bankowski Anaya HA, Shah DM, Bird IM.

Am J Physiol Heart Circ Physiol. 2013 Oct 1;305(7):H969-79. doi: 10.1152/ajpheart.00250.2013. Epub 2013 Jul 26.

10.

Regulating specific growth factor signaling using immobilized branched ligands.

Toepke MW, Impellitteri NA, Lan Levengood SK, Boeldt DS, Bird IM, Murphy WL.

Adv Healthc Mater. 2012 Jul;1(4):457-60. doi: 10.1002/adhm.201200077. Epub 2012 May 11.

12.

[Ca2+]i signaling vs. eNOS expression as determinants of NO output in uterine artery endothelium: relative roles in pregnancy adaptation and reversal by VEGF165.

Yi FX, Boeldt DS, Magness RR, Bird IM.

Am J Physiol Heart Circ Physiol. 2011 Apr;300(4):H1182-93. doi: 10.1152/ajpheart.01108.2010. Epub 2011 Jan 14.

13.

Pregnancy enhances sustained Ca2+ bursts and endothelial nitric oxide synthase activation in ovine uterine artery endothelial cells through increased connexin 43 function.

Yi FX, Boeldt DS, Gifford SM, Sullivan JA, Grummer MA, Magness RR, Bird IM.

Biol Reprod. 2010 Jan;82(1):66-75. doi: 10.1095/biolreprod.109.078253. Epub 2009 Sep 9.

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