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Items: 6

1.

Borrelia burgdorferi Manipulates Innate and Adaptive Immunity to Establish Persistence in Rodent Reservoir Hosts.

Tracy KE, Baumgarth N.

Front Immunol. 2017 Feb 20;8:116. doi: 10.3389/fimmu.2017.00116. eCollection 2017. Review.

2.

Suppression of Long-Lived Humoral Immunity Following Borrelia burgdorferi Infection.

Elsner RA, Hastey CJ, Olsen KJ, Baumgarth N.

PLoS Pathog. 2015 Jul 2;11(7):e1004976. doi: 10.1371/journal.ppat.1004976. eCollection 2015 Jul.

3.

CD4+ T cells promote antibody production but not sustained affinity maturation during Borrelia burgdorferi infection.

Elsner RA, Hastey CJ, Baumgarth N.

Infect Immun. 2015 Jan;83(1):48-56. doi: 10.1128/IAI.02471-14. Epub 2014 Oct 13.

4.

MyD88- and TRIF-independent induction of type I interferon drives naive B cell accumulation but not loss of lymph node architecture in Lyme disease.

Hastey CJ, Ochoa J, Olsen KJ, Barthold SW, Baumgarth N.

Infect Immun. 2014 Apr;82(4):1548-58. doi: 10.1128/IAI.00969-13. Epub 2014 Jan 22.

5.

Delays and diversions mark the development of B cell responses to Borrelia burgdorferi infection.

Hastey CJ, Elsner RA, Barthold SW, Baumgarth N.

J Immunol. 2012 Jun 1;188(11):5612-22. doi: 10.4049/jimmunol.1103735. Epub 2012 Apr 30.

6.

Lymphoadenopathy during lyme borreliosis is caused by spirochete migration-induced specific B cell activation.

Tunev SS, Hastey CJ, Hodzic E, Feng S, Barthold SW, Baumgarth N.

PLoS Pathog. 2011 May;7(5):e1002066. doi: 10.1371/journal.ppat.1002066. Epub 2011 May 26.

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