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Items: 4

1.

Novel Protein Kinase Inhibitors Related to Tau Pathology Modulate Tau Protein-Self Interaction Using a Luciferase Complementation Assay.

Holzer M, Schade N, Opitz A, Hilbrich I, Stieler J, Vogel T, Neukel V, Oberstadt M, Totzke F, Sch├Ąchtele C, Sippl W, Hilgeroth A.

Molecules. 2018 Sep 12;23(9). pii: E2335. doi: 10.3390/molecules23092335.

2.

BBS7 is required for BBSome formation and its absence in mice results in Bardet-Biedl syndrome phenotypes and selective abnormalities in membrane protein trafficking.

Zhang Q, Nishimura D, Vogel T, Shao J, Swiderski R, Yin T, Searby C, Carter CS, Kim G, Bugge K, Stone EM, Sheffield VC.

J Cell Sci. 2013 Jun 1;126(Pt 11):2372-80. doi: 10.1242/jcs.111740. Epub 2013 Apr 9.

3.

Bardet-Biedl syndrome 3 (Bbs3) knockout mouse model reveals common BBS-associated phenotypes and Bbs3 unique phenotypes.

Zhang Q, Nishimura D, Seo S, Vogel T, Morgan DA, Searby C, Bugge K, Stone EM, Rahmouni K, Sheffield VC.

Proc Natl Acad Sci U S A. 2011 Dec 20;108(51):20678-83. doi: 10.1073/pnas.1113220108. Epub 2011 Dec 2.

4.

Loss of earnings: a lifetime of opportunity.

Vogel T.

Med Leg J. 2002;70(Pt 4):196-7. No abstract available.

PMID:
12616793

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