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Items: 13

1.

Fibrin monomers derived from thrombogenic dysfibrinogenemia, Naples-type variant (BβAla68Thr), showed almost entirely normal polymerization.

Kamijo T, Nagata K, Taira C, Higuchi Y, Arai S, Okumura N.

Thromb Res. 2018 Dec;172:1-3. doi: 10.1016/j.thromres.2018.10.004. Epub 2018 Oct 4. No abstract available.

PMID:
30321757
2.

A Novel Mutation in the Fibrinogen Bβ Chain (c.490G>A; End of Exon 3) Causes a Splicing Abnormality and Ultimately Leads to Congenital Hypofibrinogenemia.

Taira C, Matsuda K, Arai S, Sugano M, Uehara T, Okumura N.

Int J Mol Sci. 2017 Nov 20;18(11). pii: E2470. doi: 10.3390/ijms18112470.

3.

A novel frameshift mutation in the fibrinogen γC terminal region, FGG c.1169_1170 del AT, leading to hypofibrinogenemia.

Nagata K, Arai S, Taira C, Sugano M, Honda T, Okumura N.

Thromb Res. 2017 Nov;159:82-85. doi: 10.1016/j.thromres.2017.10.002. Epub 2017 Oct 2. No abstract available.

PMID:
28992465
4.

The fibrous form of intracellular inclusion bodies in recombinant variant fibrinogen-producing cells is specific to the hepatic fibrinogen storage disease-inducible variant fibrinogen.

Arai S, Ogiwara N, Mukai S, Takezawa Y, Sugano M, Honda T, Okumura N.

Int J Hematol. 2017 Jun;105(6):758-768. doi: 10.1007/s12185-017-2185-5. Epub 2017 Feb 4.

PMID:
28161763
5.

Genetic analyses of novel compound heterozygous hypodysfibrinogenemia, Tsukuba I: FGG c.1129+62_65 del AATA and FGG c.1299+4 del A.

Mukai S, Nagata K, Ikeda M, Arai S, Sugano M, Honda T, Okumura N.

Thromb Res. 2016 Dec;148:111-117. doi: 10.1016/j.thromres.2016.11.002. Epub 2016 Nov 5.

PMID:
27837696
6.

Neutrophil left shift and white blood cell count as markers of bacterial infection.

Honda T, Uehara T, Matsumoto G, Arai S, Sugano M.

Clin Chim Acta. 2016 Jun 1;457:46-53. doi: 10.1016/j.cca.2016.03.017. Epub 2016 Mar 28. Review.

PMID:
27034055
7.

[A Case of Secondary Cryofibrinogenemia with Cholangiocarcinoma and Deep Venous Thrombosis].

Arai S, Sugano M, Honda T, Kameko F, Terasawa F, Okumura N.

Rinsho Byori. 2015 Apr;63(4):421-6. Japanese.

PMID:
26536774
8.
9.

Novel heterozygous dysfibrinogenemia, Sumida (AαC472S), showed markedly impaired lateral aggregation of protofibrils and mildly lower functional fibrinogen levels.

Ikeda M, Arai S, Mukai S, Takezawa Y, Terasawa F, Okumura N.

Thromb Res. 2015 Apr;135(4):710-7. doi: 10.1016/j.thromres.2015.01.013. Epub 2015 Jan 15.

PMID:
25613923
10.

Recombinant γT305A fibrinogen indicates severely impaired fibrin polymerization due to the aberrant function of hole 'A' and calcium binding sites.

Ikeda M, Kobayashi T, Arai S, Mukai S, Takezawa Y, Terasawa F, Okumura N.

Thromb Res. 2014 Aug;134(2):518-25. doi: 10.1016/j.thromres.2014.06.002. Epub 2014 Jun 11.

PMID:
24968960
11.

Congenital dysfibrinogenemia coincidentally diagnosed at the onset of chronic myelogenous leukemia.

Kirihara T, Fujikawa Y, Takeda W, Kurihara T, Sato K, Ueki T, Hiroshima Y, Sumi M, Ueno M, Ichikawa N, Arai S, Soya K, Okumura N, Kobayashi H.

Rinsho Ketsueki. 2014 May;55(5):541-5. Japanese.

PMID:
24881919
12.

γ375W fibrinogen-synthesizing CHO cells indicate the accumulation of variant fibrinogen within endoplasmic reticulum.

Kobayashi T, Arai S, Ogiwara N, Takezawa Y, Nanya M, Terasawa F, Okumura N.

Thromb Res. 2014 Jan;133(1):101-7. doi: 10.1016/j.thromres.2013.10.033. Epub 2013 Oct 28.

PMID:
24210681
13.

[Progress in psychosocial therapy for schizophrenic patients and their families].

Arai S.

Seishin Shinkeigaku Zasshi. 2007;109(8):743-8. Review. Japanese. No abstract available.

PMID:
18050895

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