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Items: 12

1.

Correction: Time-Dependent Progression of Demyelination and Axonal Pathology in MP4-Induced Experimental Autoimmune Encephalomyelitis.

Prinz J, Karacivi A, Stormanns ER, Recks MS, Kuerten S.

PLoS One. 2016 May 4;11(5):e0155197. doi: 10.1371/journal.pone.0155197. eCollection 2016.

2.

Time-Dependent Progression of Demyelination and Axonal Pathology in MP4-Induced Experimental Autoimmune Encephalomyelitis.

Prinz J, Karacivi A, Stormanns ER, Recks MS, Kuerten S.

PLoS One. 2015 Dec 11;10(12):e0144847. doi: 10.1371/journal.pone.0144847. eCollection 2015. Erratum in: PLoS One. 2016;11(5):e0155197.

3.

Characterization of the HCMV-Specific CD4 T Cell Responses that Are Associated with Protective Immunity.

Wunsch M, Zhang W, Hanson J, Caspell R, Karulin AY, Recks MS, Kuerten S, Sundararaman S, Lehmann PV.

Viruses. 2015 Aug 6;7(8):4414-37. doi: 10.3390/v7082828.

4.

Four different synthetic peptides of proteolipid protein induce a distinct antibody response in MP4-induced experimental autoimmune encephalomyelitis.

Recks MS, Grether NB, van der Broeck F, Ganscher A, Wagner N, Henke E, Erg√ľn S, Schroeter M, Kuerten S.

Clin Immunol. 2015 Jul;159(1):93-106. doi: 10.1016/j.clim.2015.04.020. Epub 2015 May 7.

PMID:
25959684
5.

Conventional housing conditions attenuate the development of experimental autoimmune encephalomyelitis.

Arndt A, Hoffacker P, Zellmer K, Goecer O, Recks MS, Kuerten S.

PLoS One. 2014 Jun 11;9(6):e99794. doi: 10.1371/journal.pone.0099794. eCollection 2014.

6.

Early axonal damage and progressive myelin pathology define the kinetics of CNS histopathology in a mouse model of multiple sclerosis.

Recks MS, Stormanns ER, Bader J, Arnhold S, Addicks K, Kuerten S.

Clin Immunol. 2013 Oct;149(1):32-45. doi: 10.1016/j.clim.2013.06.004. Epub 2013 Jun 18.

PMID:
23899992
7.

The complement system contributes to the pathology of experimental autoimmune encephalomyelitis by triggering demyelination and modifying the antigen-specific T and B cell response.

Hundgeburth LC, Wunsch M, Rovituso D, Recks MS, Addicks K, Lehmann PV, Kuerten S.

Clin Immunol. 2013 Mar;146(3):155-64. doi: 10.1016/j.clim.2012.12.007. Epub 2012 Dec 22.

PMID:
23352967
8.

Tertiary lymphoid organ development coincides with determinant spreading of the myelin-specific T cell response.

Kuerten S, Schickel A, Kerkloh C, Recks MS, Addicks K, Ruddle NH, Lehmann PV.

Acta Neuropathol. 2012 Dec;124(6):861-73. doi: 10.1007/s00401-012-1023-3. Epub 2012 Jul 28.

PMID:
22842876
9.

The extent of ultrastructural spinal cord pathology reflects disease severity in experimental autoimmune encephalomyelitis.

Gruppe TL, Recks MS, Addicks K, Kuerten S.

Histol Histopathol. 2012 Sep;27(9):1163-74. doi: 10.14670/HH-27.1163.

PMID:
22806903
10.

Resting of Cryopreserved PBMC Does Not Generally Benefit the Performance of Antigen-Specific T Cell ELISPOT Assays.

Kuerten S, Batoulis H, Recks MS, Karacsony E, Zhang W, Subbramanian RA, Lehmann PV.

Cells. 2012 Jul 30;1(3):409-27. doi: 10.3390/cells1030409.

11.

Experimental autoimmune encephalomyelitis--achievements and prospective advances.

Batoulis H, Recks MS, Addicks K, Kuerten S.

APMIS. 2011 Dec;119(12):819-30. doi: 10.1111/j.1600-0463.2011.02794.x. Epub 2011 Oct 18. Review.

PMID:
22085358
12.

Spinal cord histopathology of MOG peptide 35-55-induced experimental autoimmune encephalomyelitis is time- and score-dependent.

Recks MS, Addicks K, Kuerten S.

Neurosci Lett. 2011 May 2;494(3):227-31. doi: 10.1016/j.neulet.2011.03.021. Epub 2011 Mar 21.

PMID:
21406210

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