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Items: 16

1.

Understanding crash potential associated with teen driving: Survey analysis using multivariate graphical method.

Das S, Minjares-Kyle L, Wu L, Henk RH.

J Safety Res. 2019 Sep;70:213-222. doi: 10.1016/j.jsr.2019.07.009. Epub 2019 Jul 23.

PMID:
31847998
2.

Strategies to generate functionally normal neutrophils to reduce infection and infection-related mortality in cancer chemotherapy.

Abdel-Azim H, Sun W, Wu L.

Pharmacol Ther. 2019 Dec;204:107403. doi: 10.1016/j.pharmthera.2019.107403. Epub 2019 Aug 27. Review.

PMID:
31470030
3.

Examining the influence of link function misspecification in conventional regression models for developing crash modification factors.

Wu L, Lord D.

Accid Anal Prev. 2017 May;102:123-135. doi: 10.1016/j.aap.2017.02.012. Epub 2017 Mar 7.

PMID:
28282580
4.

Finite mixture modeling approach for developing crash modification factors in highway safety analysis.

Park BJ, Lord D, Wu L.

Accid Anal Prev. 2016 Dec;97:274-287. doi: 10.1016/j.aap.2016.10.023. Epub 2016 Oct 28.

PMID:
27974277
5.

Am80-GCSF synergizes myeloid expansion and differentiation to generate functional neutrophils that reduce neutropenia-associated infection and mortality.

Li L, Qi X, Sun W, Abdel-Azim H, Lou S, Zhu H, Prasadarao NV, Zhou A, Shimada H, Shudo K, Kim YM, Khazal S, He Q, Warburton D, Wu L.

EMBO Mol Med. 2016 Nov 2;8(11):1340-1359. doi: 10.15252/emmm.201606434. Print 2016 Nov.

6.

The lost intrinsic fragmentation of MAT1 protein during granulopoiesis promotes the growth and metastasis of leukemic myeloblasts.

Lou S, Liu G, Shimada H, Yang X, He Q, Wu L.

Stem Cells. 2013 Sep;31(9):1942-53. doi: 10.1002/stem.1444.

7.

Retinoid agonist Am80-enhanced neutrophil bactericidal activity arising from granulopoiesis in vitro and in a neutropenic mouse model.

Ding W, Shimada H, Li L, Mittal R, Zhang X, Shudo K, He Q, Prasadarao NV, Wu L.

Blood. 2013 Feb 7;121(6):996-1007. doi: 10.1182/blood-2012-06-436022. Epub 2012 Dec 13.

8.

Retinoid-regulated FGF8f secretion by osteoblasts bypasses retinoid stimuli to mediate granulocytic differentiation of myeloid leukemia cells.

Chaudhry P, Yang X, Wagner M, Jong A, Wu L.

Mol Cancer Ther. 2012 Feb;11(2):267-76. doi: 10.1158/1535-7163.MCT-11-0584. Epub 2011 Dec 1.

9.

Loss of CAK phosphorylation of RAR{alpha} mediates transcriptional control of retinoid-induced cancer cell differentiation.

Wang A, Alimova IN, Luo P, Jong A, Triche TJ, Wu L.

FASEB J. 2010 Mar;24(3):833-43. doi: 10.1096/fj.09-142976. Epub 2009 Nov 16.

10.

Cdc6 knockdown inhibits human neuroblastoma cell proliferation.

Feng L, Barnhart JR, Seeger RC, Wu L, Keshelava N, Huang SH, Jong A.

Mol Cell Biochem. 2008 Apr;311(1-2):189-97. doi: 10.1007/s11010-008-9709-5. Epub 2008 Feb 8.

PMID:
18259842
11.
12.

Retinoic acid induces leukemia cell G1 arrest and transition into differentiation by inhibiting cyclin-dependent kinase-activating kinase binding and phosphorylation of PML/RARalpha.

Wang JG, Barsky LW, Davicioni E, Weinberg KI, Triche TJ, Zhang XK, Wu L.

FASEB J. 2006 Oct;20(12):2142-4. Epub 2006 Aug 25.

PMID:
16935935
13.
14.

Retinoid-modulated MAT1 ubiquitination and CAK activity.

He Q, Peng H, Collins SJ, Triche TJ, Wu L.

FASEB J. 2004 Nov;18(14):1734-6. Epub 2004 Sep 2.

PMID:
15345685
15.

MAT1-modulated cyclin-dependent kinase-activating kinase activity cross-regulates neuroblastoma cell G1 arrest and neurite outgrowth.

Zhang S, He Q, Peng H, Tedeschi-Blok N, Triche TJ, Wu L.

Cancer Res. 2004 May 1;64(9):2977-83.

16.

Retinoid-induced G1 arrest and differentiation activation are associated with a switch to cyclin-dependent kinase-activating kinase hypophosphorylation of retinoic acid receptor alpha.

Wang J, Barsky LW, Shum CH, Jong A, Weinberg KI, Collins SJ, Triche TJ, Wu L.

J Biol Chem. 2002 Nov 8;277(45):43369-76. Epub 2002 Sep 3.

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