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Items: 19

1.

2-Deoxyglucose and Beta-Hydroxybutyrate: Metabolic Agents for Seizure Control.

Rho JM, Shao LR, Stafstrom CE.

Front Cell Neurosci. 2019 Apr 30;13:172. doi: 10.3389/fncel.2019.00172. eCollection 2019. Review.

2.

Pediatric Epilepsy Mechanisms: Expanding the Paradigm of Excitation/Inhibition Imbalance.

Shao LR, Habela CW, Stafstrom CE.

Children (Basel). 2019 Feb 5;6(2). pii: E23. doi: 10.3390/children6020023. Review.

3.

Glycolytic inhibition: A novel approach toward controlling neuronal excitability and seizures.

Shao LR, Rho JM, Stafstrom CE.

Epilepsia Open. 2018 Aug 19;3(Suppl Suppl 2):191-197. doi: 10.1002/epi4.12251. eCollection 2018 Dec.

4.

The Glycolytic Metabolite, Fructose-1,6-bisphosphate, Blocks Epileptiform Bursts by Attenuating Voltage-Activated Calcium Currents in Hippocampal Slices.

Shao LR, Wang G, Stafstrom CE.

Front Cell Neurosci. 2018 Jun 15;12:168. doi: 10.3389/fncel.2018.00168. eCollection 2018.

5.

Glycolytic inhibition by 2-deoxy-d-glucose abolishes both neuronal and network bursts in an in vitro seizure model.

Shao LR, Stafstrom CE.

J Neurophysiol. 2017 Jul 1;118(1):103-113. doi: 10.1152/jn.00100.2017. Epub 2017 Apr 12.

6.

Pediatric Epileptic Encephalopathies: Pathophysiology and Animal Models.

Shao LR, Stafstrom CE.

Semin Pediatr Neurol. 2016 May;23(2):98-107. doi: 10.1016/j.spen.2016.05.004. Epub 2016 May 24. Review.

PMID:
27544466
7.

Synaptic and extrasynaptic plasticity in glutamatergic circuits involving dentate granule cells following chronic N-methyl-D-aspartate receptor inhibition.

He S, Shao LR, Wang Y, Bausch SB.

J Neurophysiol. 2013 Mar;109(6):1535-47. doi: 10.1152/jn.00667.2012. Epub 2012 Dec 19.

8.

Increased Kv1 channel expression may contribute to decreased sIPSC frequency following chronic inhibition of NR2B-containing NMDAR.

He S, Shao LR, Rittase WB, Bausch SB.

Neuropsychopharmacology. 2012 May;37(6):1338-56. doi: 10.1038/npp.2011.320. Epub 2012 Jan 4.

9.

Repetitive perforant-path stimulation induces epileptiform bursts in minislices of dentate gyrus from rats with kainate-induced epilepsy.

Shao LR, Dudek FE.

J Neurophysiol. 2011 Feb;105(2):522-7. doi: 10.1152/jn.00456.2010. Epub 2010 Dec 8.

10.
11.

Changes in mIPSCs and sIPSCs after kainate treatment: status epilepticus-induced neuronal loss or direct activation of kainate receptors?

Shao LR, Dudek FE.

J Neurophysiol. 2006 Aug;96(2):961-2. Epub 2006 May 3. No abstract available.

12.

Mossy fiber sprouting and recurrent excitation: direct electrophysiologic evidence and potential implications.

Dudek FE, Shao LR.

Epilepsy Curr. 2004 Sep-Oct;4(5):184-7. No abstract available.

14.

Loss of GABAergic Interneurons in Seizure-induced Epileptogenesis.

Dudek FE, Shao LR.

Epilepsy Curr. 2003 Sep;3(5):159-161. No abstract available.

17.

Synaptic Reorganization in an Experimental Model of Posttraumatic Epilepsy.

Dudek FE, Shao LR.

Epilepsy Curr. 2002 Nov;2(6):198-199. No abstract available.

18.
19.

Riluzole-sensitive slowly inactivating sodium current in rat suprachiasmatic nucleus neurons.

Kononenko NI, Shao LR, Dudek FE.

J Neurophysiol. 2004 Feb;91(2):710-8. Epub 2003 Oct 22.

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