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Items: 16


Estrogen amplifies pre-existing atopy-associated Th2-bias in an experimental asthma model.

Lauzon-Joset JF, Mincham KT, Abad AP, Short BP, Holt PG, Strickland DH, Leffler J.

Clin Exp Allergy. 2019 Nov 30. doi: 10.1111/cea.13544. [Epub ahead of print]


Progressive increase of FcεRI expression across several PBMC subsets is associated with atopy and atopic asthma within school-aged children.

Leffler J, Read JF, Jones AC, Mok D, Hollams EM, Laing IA, Le Souef PN, Sly PD, Kusel MMH, de Klerk NH, Bosco A, Holt PG, Strickland DH.

Pediatr Allergy Immunol. 2019 Sep;30(6):646-653. doi: 10.1111/pai.13063. Epub 2019 May 22.


Transplacental immune modulation with a bacterial-derived agent protects against allergic airway inflammation.

Mincham KT, Scott NM, Lauzon-Joset JF, Leffler J, Larcombe AN, Stumbles PA, Robertson SA, Pasquali C, Holt PG, Strickland DH.

J Clin Invest. 2018 Nov 1;128(11):4856-4869. doi: 10.1172/JCI122631. Epub 2018 Oct 2.


Basophil counts in PBMC populations during childhood acute wheeze/asthma are associated with future exacerbations.

Leffler J, Jones AC, Hollams EM, Prastanti F, Le Souëf PN, Holt PG, Bosco A, Laing IA, Strickland DH.

J Allergy Clin Immunol. 2018 Nov;142(5):1639-1641.e5. doi: 10.1016/j.jaci.2018.07.003. Epub 2018 Jul 21. No abstract available.


Immunological Processes Driving IgE Sensitisation and Disease Development in Males and Females.

Leffler J, Stumbles PA, Strickland DH.

Int J Mol Sci. 2018 May 23;19(6). pii: E1554. doi: 10.3390/ijms19061554. Review.


Functional differences in airway dendritic cells determine susceptibility to IgE-sensitization.

Leffler J, Mincham KT, Mok D, Blank F, Holt PG, Stumbles PA, Strickland DH.

Immunol Cell Biol. 2018 Mar;96(3):316-329. doi: 10.1111/imcb.12005. Epub 2018 Feb 2.


Plasma C4d as marker for lupus nephritis in systemic lupus erythematosus.

Martin M, Smoląg KI, Björk A, Gullstrand B, Okrój M, Leffler J, Jönsen A, Bengtsson AA, Blom AM.

Arthritis Res Ther. 2017 Dec 6;19(1):266. doi: 10.1186/s13075-017-1470-2.


Decreased Neutrophil Extracellular Trap Degradation in Shiga Toxin-Associated Haemolytic Uraemic Syndrome.

Leffler J, Prohászka Z, Mikes B, Sinkovits G, Ciacma K, Farkas P, Réti M, Kelen K, Reusz GS, Szabó AJ, Martin M, Blom AM.

J Innate Immun. 2017;9(1):12-21. doi: 10.1159/000450609. Epub 2016 Oct 27.


Factor H Autoantibodies in Patients with Antiphospholipid Syndrome and Thrombosis.

Foltyn Zadura A, Memon AA, Stojanovich L, Perricone C, Conti F, Valesini G, Bogdanovic G, Hillarp A, Shoenfeld Y, Sundquist J, Leffler J, Svensson PJ, Trouw LA, Blom AM.

J Rheumatol. 2015 Oct;42(10):1786-93. doi: 10.3899/jrheum.150185. Epub 2015 Aug 15.


A subset of patients with systemic lupus erythematosus fails to degrade DNA from multiple clinically relevant sources.

Leffler J, Ciacma K, Gullstrand B, Bengtsson AA, Martin M, Blom AM.

Arthritis Res Ther. 2015 Aug 13;17:205. doi: 10.1186/s13075-015-0726-y.


The complement system in systemic lupus erythematosus: an update.

Leffler J, Bengtsson AA, Blom AM.

Ann Rheum Dis. 2014 Sep;73(9):1601-6. doi: 10.1136/annrheumdis-2014-205287. Epub 2014 May 20. Review.


Degradation of neutrophil extracellular traps is decreased in patients with antiphospholipid syndrome.

Leffler J, Stojanovich L, Shoenfeld Y, Bogdanovic G, Hesselstrand R, Blom AM.

Clin Exp Rheumatol. 2014 Jan-Feb;32(1):66-70. Epub 2013 Dec 2.


Degradation of neutrophil extracellular traps co-varies with disease activity in patients with systemic lupus erythematosus.

Leffler J, Gullstrand B, Jönsen A, Nilsson JÅ, Martin M, Blom AM, Bengtsson AA.

Arthritis Res Ther. 2013 Aug 14;15(4):R84. doi: 10.1186/ar4264.


Annexin A2 and A5 serve as new ligands for C1q on apoptotic cells.

Martin M, Leffler J, Blom AM.

J Biol Chem. 2012 Sep 28;287(40):33733-44. Epub 2012 Aug 9.


Neutrophil extracellular traps that are not degraded in systemic lupus erythematosus activate complement exacerbating the disease.

Leffler J, Martin M, Gullstrand B, Tydén H, Lood C, Truedsson L, Bengtsson AA, Blom AM.

J Immunol. 2012 Apr 1;188(7):3522-31. doi: 10.4049/jimmunol.1102404. Epub 2012 Feb 17.


Annexin-II, DNA, and histones serve as factor H ligands on the surface of apoptotic cells.

Leffler J, Herbert AP, Norström E, Schmidt CQ, Barlow PN, Blom AM, Martin M.

J Biol Chem. 2010 Feb 5;285(6):3766-76. doi: 10.1074/jbc.M109.045427. Epub 2009 Dec 1.

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