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Items: 5

1.

GalK limits type I-F CRISPR-Cas expression in a CRP-dependent manner.

Hampton HG, Patterson AG, Chang JT, Taylor C, Fineran PC.

FEMS Microbiol Lett. 2019 Jun 1;366(11). pii: fnz137. doi: 10.1093/femsle/fnz137.

PMID:
31226710
2.

Regulation of the Type I-F CRISPR-Cas system by CRP-cAMP and GalM controls spacer acquisition and interference.

Patterson AG, Chang JT, Taylor C, Fineran PC.

Nucleic Acids Res. 2015 Jul 13;43(12):6038-48. doi: 10.1093/nar/gkv517. Epub 2015 May 24.

3.

Priming in the Type I-F CRISPR-Cas system triggers strand-independent spacer acquisition, bi-directionally from the primed protospacer.

Richter C, Dy RL, McKenzie RE, Watson BN, Taylor C, Chang JT, McNeil MB, Staals RH, Fineran PC.

Nucleic Acids Res. 2014 Jul;42(13):8516-26. doi: 10.1093/nar/gku527. Epub 2014 Jul 2.

4.

Cytotoxic chromosomal targeting by CRISPR/Cas systems can reshape bacterial genomes and expel or remodel pathogenicity islands.

Vercoe RB, Chang JT, Dy RL, Taylor C, Gristwood T, Clulow JS, Richter C, Przybilski R, Pitman AR, Fineran PC.

PLoS Genet. 2013 Apr;9(4):e1003454. doi: 10.1371/journal.pgen.1003454. Epub 2013 Apr 18.

5.

Function and regulation of clustered regularly interspaced short palindromic repeats (CRISPR) / CRISPR associated (Cas) systems.

Richter C, Chang JT, Fineran PC.

Viruses. 2012 Oct 19;4(10):2291-311. doi: 10.3390/v4102291. Review.

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