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Items: 15

1.

Linking the Endoplasmic Reticulum to Parkinson's Disease and Alpha-Synucleinopathy.

Colla E.

Front Neurosci. 2019 May 29;13:560. doi: 10.3389/fnins.2019.00560. eCollection 2019. Review.

2.

Microbiome, Parkinson's Disease and Molecular Mimicry.

Miraglia F, Colla E.

Cells. 2019 Mar 7;8(3). pii: E222. doi: 10.3390/cells8030222. Review.

3.

Constipation, deficit in colon contractions and alpha-synuclein inclusions within the colon precede motor abnormalities and neurodegeneration in the central nervous system in a mouse model of alpha-synucleinopathy.

Rota L, Pellegrini C, Benvenuti L, Antonioli L, Fornai M, Blandizzi C, Cattaneo A, Colla E.

Transl Neurodegener. 2019 Feb 6;8:5. doi: 10.1186/s40035-019-0146-z. eCollection 2019.

4.

Subcellular localization of alpha-synuclein aggregates and their interaction with membranes.

Miraglia F, Ricci A, Rota L, Colla E.

Neural Regen Res. 2018 Jul;13(7):1136-1144. doi: 10.4103/1673-5374.235013. Review.

5.
6.

Toxic properties of microsome-associated alpha-synuclein species in mouse primary neurons.

Colla E, Panattoni G, Ricci A, Rizzi C, Rota L, Carucci N, Valvano V, Gobbo F, Capsoni S, Lee MK, Cattaneo A.

Neurobiol Dis. 2018 Mar;111:36-47. doi: 10.1016/j.nbd.2017.12.004. Epub 2017 Dec 12.

7.

Endoplasmic reticulum stress is important for the manifestations of α-synucleinopathy in vivo.

Colla E, Coune P, Liu Y, Pletnikova O, Troncoso JC, Iwatsubo T, Schneider BL, Lee MK.

J Neurosci. 2012 Mar 7;32(10):3306-20. doi: 10.1523/JNEUROSCI.5367-11.2012.

8.

Accumulation of toxic α-synuclein oligomer within endoplasmic reticulum occurs in α-synucleinopathy in vivo.

Colla E, Jensen PH, Pletnikova O, Troncoso JC, Glabe C, Lee MK.

J Neurosci. 2012 Mar 7;32(10):3301-5. doi: 10.1523/JNEUROSCI.5368-11.2012.

9.

Regulation of neuronal survival factor MEF2D by chaperone-mediated autophagy.

Yang Q, She H, Gearing M, Colla E, Lee M, Shacka JJ, Mao Z.

Science. 2009 Jan 2;323(5910):124-7. doi: 10.1126/science.1166088.

10.

Novel nuclear localization signal regulated by ambient tonicity in vertebrates.

Kwon MS, Lee SD, Kim JA, Colla E, Choi YJ, Suh PG, Kwon HM.

J Biol Chem. 2008 Aug 15;283(33):22400-9. doi: 10.1074/jbc.M710550200. Epub 2008 Jun 25.

11.

Wild-type and mutant alpha-synuclein induce a multi-component gene expression profile consistent with shared pathophysiology in different transgenic mouse models of PD.

Miller RM, Kiser GL, Kaysser-Kranich T, Casaceli C, Colla E, Lee MK, Palaniappan C, Federoff HJ.

Exp Neurol. 2007 Mar;204(1):421-32. Epub 2007 Jan 24.

PMID:
17254569
12.

TonEBP is inhibited by RNA helicase A via interaction involving the E'F loop.

Colla E, Lee SD, Sheen MR, Woo SK, Kwon HM.

Biochem J. 2006 Jan 1;393(Pt 1):411-9.

13.

Aggregation promoting C-terminal truncation of alpha-synuclein is a normal cellular process and is enhanced by the familial Parkinson's disease-linked mutations.

Li W, West N, Colla E, Pletnikova O, Troncoso JC, Marsh L, Dawson TM, Jäkälä P, Hartmann T, Price DL, Lee MK.

Proc Natl Acad Sci U S A. 2005 Feb 8;102(6):2162-7. Epub 2005 Jan 31.

14.

Multiple domains of TonEBP cooperate to stimulate transcription in response to hypertonicity.

Lee SD, Colla E, Sheen MR, Na KY, Kwon HM.

J Biol Chem. 2003 Nov 28;278(48):47571-7. Epub 2003 Sep 11.

15.

Two-way arginine transport in human endothelial cells: TNF-alpha stimulation is restricted to system y(+).

Sala R, Rotoli BM, Colla E, Visigalli R, Parolari A, Bussolati O, Gazzola GC, Dall'Asta V.

Am J Physiol Cell Physiol. 2002 Jan;282(1):C134-43.

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