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Items: 1 to 20 of 52

1.

Alpha-Tocotrienol Prevents Oxidative Stress-Mediated Post-Translational Cleavage of Bcl-xL in Primary Hippocampal Neurons.

Park HA, Mnatsakanyan N, Broman K, Davis AU, May J, Licznerski P, Crowe-White KM, Lackey KH, Jonas EA.

Int J Mol Sci. 2019 Dec 28;21(1). pii: E220. doi: 10.3390/ijms21010220.

2.

A mitochondrial megachannel resides in monomeric F1FO ATP synthase.

Mnatsakanyan N, Llaguno MC, Yang Y, Yan Y, Weber J, Sigworth FJ, Jonas EA.

Nat Commun. 2019 Dec 20;10(1):5823. doi: 10.1038/s41467-019-13766-2.

3.

Dopamine fuels its own release.

Chen R, Jonas EA.

Nat Neurosci. 2020 Jan;23(1):1-2. doi: 10.1038/s41593-019-0563-4. No abstract available.

PMID:
31844312
4.

Parkinson's disease protein DJ-1 regulates ATP synthase protein components to increase neuronal process outgrowth.

Chen R, Park HA, Mnatsakanyan N, Niu Y, Licznerski P, Wu J, Miranda P, Graham M, Tang J, Boon AJW, Cossu G, Mandemakers W, Bonifati V, Smith PJS, Alavian KN, Jonas EA.

Cell Death Dis. 2019 Jun 13;10(6):469. doi: 10.1038/s41419-019-1679-x.

5.

The mitochondrial metabolic function of DJ-1 is modulated by 14-3-3β.

Weinert M, Millet A, Jonas EA, Alavian KN.

FASEB J. 2019 Aug;33(8):8925-8934. doi: 10.1096/fj.201802754R. Epub 2019 Apr 29.

6.

ATP Synthase C-Subunit-Deficient Mitochondria Have a Small Cyclosporine A-Sensitive Channel, but Lack the Permeability Transition Pore.

Neginskaya MA, Solesio ME, Berezhnaya EV, Amodeo GF, Mnatsakanyan N, Jonas EA, Pavlov EV.

Cell Rep. 2019 Jan 2;26(1):11-17.e2. doi: 10.1016/j.celrep.2018.12.033.

7.

Nutritional Regulators of Bcl-xL in the Brain.

Park HA, Broman K, Stumpf A, Kazyak S, Jonas EA.

Molecules. 2018 Nov 19;23(11). pii: E3019. doi: 10.3390/molecules23113019. Review.

8.

Cardiac metabolic effects of KNa1.2 channel deletion and evidence for its mitochondrial localization.

Smith CO, Wang YT, Nadtochiy SM, Miller JH, Jonas EA, Dirksen RT, Nehrke K, Brookes PS.

FASEB J. 2018 Jun 4:fj201800139R. doi: 10.1096/fj.201800139R. [Epub ahead of print]

9.

BDNF signaling: Harnessing stress to battle mood disorder.

Licznerski P, Jonas EA.

Proc Natl Acad Sci U S A. 2018 Apr 10;115(15):3742-3744. doi: 10.1073/pnas.1803645115. Epub 2018 Mar 28. No abstract available.

10.

ΔN-Bcl-xL, a therapeutic target for neuroprotection.

Park HA, Jonas EA.

Neural Regen Res. 2017 Nov;12(11):1791-1794. doi: 10.4103/1673-5374.219033. Review.

11.

Guidelines on experimental methods to assess mitochondrial dysfunction in cellular models of neurodegenerative diseases.

Connolly NMC, Theurey P, Adam-Vizi V, Bazan NG, Bernardi P, Bolaños JP, Culmsee C, Dawson VL, Deshmukh M, Duchen MR, Düssmann H, Fiskum G, Galindo MF, Hardingham GE, Hardwick JM, Jekabsons MB, Jonas EA, Jordán J, Lipton SA, Manfredi G, Mattson MP, McLaughlin B, Methner A, Murphy AN, Murphy MP, Nicholls DG, Polster BM, Pozzan T, Rizzuto R, Satrústegui J, Slack RS, Swanson RA, Swerdlow RH, Will Y, Ying Z, Joselin A, Gioran A, Moreira Pinho C, Watters O, Salvucci M, Llorente-Folch I, Park DS, Bano D, Ankarcrona M, Pizzo P, Prehn JHM.

Cell Death Differ. 2018 Mar;25(3):542-572. doi: 10.1038/s41418-017-0020-4. Epub 2017 Dec 11. Review.

12.

Phylogenetic Profiling of Mitochondrial Proteins and Integration Analysis of Bacterial Transcription Units Suggest Evolution of F1Fo ATP Synthase from Multiple Modules.

Niu Y, Moghimyfiroozabad S, Safaie S, Yang Y, Jonas EA, Alavian KN.

J Mol Evol. 2017 Dec;85(5-6):219-233. doi: 10.1007/s00239-017-9819-3. Epub 2017 Nov 24.

13.

Inhibition of Bcl-xL prevents pro-death actions of ΔN-Bcl-xL at the mitochondrial inner membrane during glutamate excitotoxicity.

Park HA, Licznerski P, Mnatsakanyan N, Niu Y, Sacchetti S, Wu J, Polster BM, Alavian KN, Jonas EA.

Cell Death Differ. 2017 Nov;24(11):1963-1974. doi: 10.1038/cdd.2017.123. Epub 2017 Aug 4.

14.

Neuronal Death After Hemorrhagic Stroke In Vitro and In Vivo Shares Features of Ferroptosis and Necroptosis.

Zille M, Karuppagounder SS, Chen Y, Gough PJ, Bertin J, Finger J, Milner TA, Jonas EA, Ratan RR.

Stroke. 2017 Apr;48(4):1033-1043. doi: 10.1161/STROKEAHA.116.015609. Epub 2017 Mar 1.

15.

Erratum to: The Mitochondrial Permeability Transition Pore and ATP Synthase.

Beutner G, Alavian KN, Jonas EA, Porter GA Jr.

Handb Exp Pharmacol. 2017;240:489. doi: 10.1007/164_2016_87. No abstract available.

PMID:
27913888
16.

The Mitochondrial Permeability Transition Pore and ATP Synthase.

Beutner G, Alavian KN, Jonas EA, Porter GA Jr.

Handb Exp Pharmacol. 2017;240:21-46. doi: 10.1007/164_2016_5. Erratum in: Handb Exp Pharmacol. 2017;240:489.

PMID:
27590224
17.

Physiological roles of the mitochondrial permeability transition pore.

Mnatsakanyan N, Beutner G, Porter GA, Alavian KN, Jonas EA.

J Bioenerg Biomembr. 2017 Feb;49(1):13-25. doi: 10.1007/s10863-016-9652-1. Epub 2016 Feb 11. Review.

18.

Decreased SGK1 Expression and Function Contributes to Behavioral Deficits Induced by Traumatic Stress.

Licznerski P, Duric V, Banasr M, Alavian KN, Ota KT, Kang HJ, Jonas EA, Ursano R, Krystal JH, Duman RS; Traumatic Stress Brain Study Group.

PLoS Biol. 2015 Oct 27;13(10):e1002282. doi: 10.1371/journal.pbio.1002282. eCollection 2015 Oct.

19.

Cell death disguised: The mitochondrial permeability transition pore as the c-subunit of the F(1)F(O) ATP synthase.

Jonas EA, Porter GA Jr, Beutner G, Mnatsakanyan N, Alavian KN.

Pharmacol Res. 2015 Sep;99:382-92. doi: 10.1016/j.phrs.2015.04.013. Epub 2015 May 5. Review.

20.

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