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Items: 5

1.

Notch and Wnt Dysregulation and Its Relevance for Breast Cancer and Tumor Initiation.

Braune EB, Seshire A, Lendahl U.

Biomedicines. 2018 Nov 1;6(4). pii: E101. doi: 10.3390/biomedicines6040101. Review.

2.

Notch signaling promotes a HIF2α-driven hypoxic response in multiple tumor cell types.

Mutvei AP, Landor SK, Fox R, Braune EB, Tsoi YL, Phoon YP, Sahlgren C, Hartman J, Bergh J, Jin S, Lendahl U.

Oncogene. 2018 Nov;37(46):6083-6095. doi: 10.1038/s41388-018-0400-3. Epub 2018 Jul 11.

3.

Notch -- a goldilocks signaling pathway in disease and cancer therapy.

Braune EB, Lendahl U.

Discov Med. 2016 Mar;21(115):189-96. Review.

4.

Loss of CSL Unlocks a Hypoxic Response and Enhanced Tumor Growth Potential in Breast Cancer Cells.

Braune EB, Tsoi YL, Phoon YP, Landor S, Silva Cascales H, Ramsköld D, Deng Q, Lindqvist A, Lian X, Sahlgren C, Jin SB, Lendahl U.

Stem Cell Reports. 2016 May 10;6(5):643-651. doi: 10.1016/j.stemcr.2016.03.004. Epub 2016 Apr 7.

5.

S/T phosphorylation of DLL1 is required for full ligand activity in vitro but dispensable for DLL1 function in vivo during embryonic patterning and marginal zone B cell development.

Braune EB, Schuster-Gossler K, Lyszkiewicz M, Serth K, Preusse K, Madlung J, Macek B, Krueger A, Gossler A.

Mol Cell Biol. 2014 Apr;34(7):1221-33. doi: 10.1128/MCB.00965-13. Epub 2014 Jan 21.

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