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Items: 10

1.

Mitochondrial bioenergetic changes during development as an indicator of C. elegans health-span.

Maglioni S, Mello DF, Schiavi A, Meyer JN, Ventura N.

Aging (Albany NY). 2019 Aug 27;11(16):6535-6554. doi: 10.18632/aging.102208. Epub 2019 Aug 27.

2.

MANF deletion abrogates early larval Caenorhabditis elegans stress response to tunicamycin and Pseudomonas aeruginosa.

Hartman JH, Richie CT, Gordon KL, Mello DF, Castillo P, Zhu A, Wang Y, Hoffer BJ, Sherwood DR, Meyer JN, Harvey BK.

Eur J Cell Biol. 2019 May 21. pii: S0171-9335(18)30348-0. doi: 10.1016/j.ejcb.2019.05.002. [Epub ahead of print]

PMID:
31138438
3.
4.

Two epizootic Perkinsus spp. events in commercial oyster farms at Santa Catarina, Brazil.

Luz Cunha AC, Pontinha VA, de Castro MAM, Sühnel S, Medeiros SC, Moura da Luz ÂM, Harakava R, Tachibana L, Mello DF, Danielli NM, Dafre AL, Magalhães ARM, P Mouriño JL.

J Fish Dis. 2019 Mar;42(3):455-463. doi: 10.1111/jfd.12958. Epub 2019 Jan 18.

PMID:
30659615
5.

Nonselective autophagy reduces mitochondrial content during starvation in Caenorhabditis elegans.

Hibshman JD, Leuthner TC, Shoben C, Mello DF, Sherwood DR, Meyer JN, Baugh LR.

Am J Physiol Cell Physiol. 2018 Dec 1;315(6):C781-C792. doi: 10.1152/ajpcell.00109.2018. Epub 2018 Aug 22.

PMID:
30133321
6.

Mitochondrial Toxicity.

Meyer JN, Hartman JH, Mello DF.

Toxicol Sci. 2018 Mar 1;162(1):15-23. doi: 10.1093/toxsci/kfy008. Review.

7.

Thiol oxidation of hemolymph proteins in oysters Crassostrea brasiliana as markers of oxidative damage induced by urban sewage exposure.

Trevisan R, Flores-Nunes F, Dolores ES, Mattos JJ, Piazza CE, Sasaki ST, Taniguchi S, Montone RC, Bícego MC, Dos Reis IMM, Zacchi FL, Othero BNM, Bastolla CLV, Mello DF, Fraga APM, Wendt N, Toledo-Silva G, Razzera G, Dafre AL, de Melo CMR, Bianchini A, Marques MRF, Bainy ACD.

Environ Toxicol Chem. 2017 Jul;36(7):1833-1845. doi: 10.1002/etc.3543. Epub 2016 Aug 12.

PMID:
27363828
8.

Gills as a glutathione-dependent metabolic barrier in Pacific oysters Crassostrea gigas: Absorption, metabolism and excretion of a model electrophile.

Trevisan R, Mello DF, Delapedra G, Silva DGH, Arl M, Danielli NM, Metian M, Almeida EA, Dafre AL.

Aquat Toxicol. 2016 Apr;173:105-119. doi: 10.1016/j.aquatox.2016.01.008. Epub 2016 Jan 29.

PMID:
26859778
9.

Gills are an initial target of zinc oxide nanoparticles in oysters Crassostrea gigas, leading to mitochondrial disruption and oxidative stress.

Trevisan R, Delapedra G, Mello DF, Arl M, Schmidt ÉC, Meder F, Monopoli M, Cargnin-Ferreira E, Bouzon ZL, Fisher AS, Sheehan D, Dafre AL.

Aquat Toxicol. 2014 Aug;153:27-38. doi: 10.1016/j.aquatox.2014.03.018. Epub 2014 Apr 1.

PMID:
24745718
10.

Cellular and transcriptional responses of Crassostrea gigas hemocytes exposed in vitro to brevetoxin (PbTx-2).

Mello DF, de Oliveira ES, Vieira RC, Simoes E, Trevisan R, Dafre AL, Barracco MA.

Mar Drugs. 2012 Mar;10(3):583-97. doi: 10.3390/md10030583. Epub 2012 Mar 5.

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