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Trends Microbiol. 1996 Jan;4(1):38-42.

Regulation of Epstein-Barr virus latency by latent membrane protein 2.

Author information

1
Microbiology-Immunology Dept, Northwestern University Medical School, Chicago, IL 60611, USA. r-longnecker@nwu.edu

Abstract

Like other herpesviruses, Epstein-Barr virus persists in its host through its ability to establish a latent infection that periodically reactivates. Latent membrane protein 2A (LMP2A) regulates reactivation from latency by interfering with normal B cell signal transduction processes, and may define a new class of regulators of herpesvirus latency.

PMID:
8824794
[Indexed for MEDLINE]

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