Format

Send to

Choose Destination
Cell Death Dis. 2018 Jul 23;9(8):800. doi: 10.1038/s41419-018-0773-9.

Correction: Increased survival and cell cycle progression pathways are required for EWS/FLI1-induced malignant transformation.

Author information

1
Ludwig Boltzmann Institute for Cancer Research, Vienna, Austria.
2
Institute of Animal Breeding and Genetics, University of Veterinary Medicine, Vienna, Austria.
3
Medical University of Vienna, Vienna, Austria.
4
Center of Physiology and Pharmacology, Vienna, Austria.
5
Clinical Institute of Pathology, Vienna, Austria.
6
Children's Cancer Research Institute, St. Anna Kinderkrebsforschung, Vienna, Austria.
7
CeMM Research Center for Molecular Medicine of the Austrian Academy of Sciences, Vienna, Austria.
8
Department of Pediatrics, Medical University of Vienna, Vienna, Austria.
9
Randall Division of Cell and Molecular Biophysics, King's College London, London, UK.
10
Department of Bone and Skeletal Research, Institute of Experimental Musculoskeletal Medicine, University Hospital Münster, Münster, Germany.
11
Institute of General Zoology and Endocrinology, University of Ulm, Ulm, Germany.
12
University of Veterinary Medicine, Vienna, Austria.
13
Institute of Medical Genetics, Vienna, Austria.
14
Georgetown University Medical Center, Lombardi Comprehensive Cancer Center, Washington, USA.
15
Children's Cancer Research Centre and Department of Pediatrics, Klinikum rechts der Isar, Technische Universität München, Munich, Germany.
16
Ludwig Boltzmann Institute for Cancer Research, Vienna, Austria. richard.moriggl@lbicr.lbg.ac.at.
17
Institute of Animal Breeding and Genetics, University of Veterinary Medicine, Vienna, Austria. richard.moriggl@lbicr.lbg.ac.at.
18
Medical University of Vienna, Vienna, Austria. richard.moriggl@lbicr.lbg.ac.at.

Abstract

Since publication of the article, the authors became aware that Figure 6(A,D) contained errors in the bands and loading controls. The newly compiled Figure 6A and 6D is given below.

Supplemental Content

Full text links

Icon for Nature Publishing Group Icon for PubMed Central
Loading ...
Support Center