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Clin Immunol. 2017 Oct;183:263-265. doi: 10.1016/j.clim.2017.08.020. Epub 2017 Sep 4.

DOCK8 and STAT3 dependent inhibition of IgE isotype switching by TLR9 ligation in human B cells.

Author information

1
Boston Children's Hospital, Harvard Medical School, Boston, MA 02115, USA.
2
Department of Pediatrics, Faculty of Medicine, Kuwait University, and Allergy and Clinical Immunology Unit, Department of Pediatrics, Al-Sabah Hospital, Kuwait.
3
Institut de Génétique Humaine, UMR 9002 CNRS-Université de Montpellier, Montpellier, France.
4
National Institutes of Health, Bethesda, MD 20892, USA.
5
Division of Pediatric Allergy and Immunology, Meram Medical Faculty, Necmettin Erbakan University, Konya, Turkey.
6
Department of Pediatric Allergy and Immunology, Ankara Children's Hematology Oncology Training and Research Hospital, Ankara, Turkey.
7
Department of Pediatrics, and Department of Internal Medicine, Division of Genetics, Endocrinology & Metabolism, University of Kansas Medical Center, Kansas City, KS 66215, USA.
8
Department of Pediatrics, Mansoura University Children's Hospital, Faculty of Medicine, Mansoura University, Egypt.
9
Department of Immunology-Histocompatibility, "Aghia Sophia" Children's Hospital, Athens, Greece.
10
Department of Pediatrics, Allergy and Clinical Immunology Unit, Royal Hospital, Muscat, Oman.
11
Faculty of Medicine, Division of Pediatric Allergy and Immunology, Marmara University, Istanbul, Turkey.
12
Department of Pediatrics, University of Washington, and Seattle Children's Research Institute, Seattle, WA 98101, USA.
13
Division of Transfusion Medicine, Department of Pathology, Harvard Medical School, Boston, MA 02115, USA.
14
Boston Children's Hospital, Harvard Medical School, Boston, MA 02115, USA. Electronic address: raif.geha@childrens.harvard.edu.

KEYWORDS:

CpG; DOCK8; Hyper IgE Syndrome; IgE synthesis; STAT3

PMID:
28882618
DOI:
10.1016/j.clim.2017.08.020
[Indexed for MEDLINE]

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