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Cell Host Microbe. 2016 Jul 13;20(1):9-11. doi: 10.1016/j.chom.2016.06.013.

Placental Mechanics in the Zika-Microcephaly Relationship.

Author information

1
Department of Epidemiology, University of Pittsburgh, Pittsburgh, PA 15261, USA; Department of Obstetrics, Gynecology, and Reproductive Sciences, University of Pittsburgh, Pittsburgh, PA 15261, USA. Electronic address: adibij@pitt.edu.
2
Department of Epidemiology, University of Pittsburgh, Pittsburgh, PA 15261, USA.
3
Department of Infectious Diseases and Microbiology, University of Pittsburgh, Pittsburgh, PA 15261, USA.
4
Department of Bioengineering, University of Pittsburgh, Pittsburgh, PA 15261, USA; Department of Developmental Biology, University of Pittsburgh, Pittsburgh, PA 15261, USA; Department of Computational and Systems Biology, University of Pittsburgh, Pittsburgh, PA 15261, USA.

Abstract

How the Zika virus (ZIKV) accesses the embryo remains unknown. In this issue, Quicke et al. (2016) use an in vitro model of the human placenta to show that placental macrophages are more permissive to ZIKV infection than trophoblasts, which may be refractory to infection (Bayer et al., 2016).

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PMID:
27414496
DOI:
10.1016/j.chom.2016.06.013
[Indexed for MEDLINE]
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