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J Invest Dermatol. 2016 Mar;136(3):559-60. doi: 10.1016/j.jid.2015.11.016.

Staphylococcus epidermidis Sets Things Right Again.

Author information

1
Department of Dermatology and Allergology, Technical University Munich, Munich, Germany.
2
Department of Dermatology and Allergology, Technical University Munich, Munich, Germany. Electronic address: tilo.biedermann@tum.de.

Abstract

Commensal cutaneous bacteria are believed to play a role in skin homeostasis, possibly by counteracting the effects of pathogenic inflammation. In this issue, Xia et al. show that Staphylococcus epidermidis is involved in the regulation of Propionibacterium acnes-induced inflammation. Staphylococcal lipoteichoic acid induces miR-143, which in turn inhibits toll-like receptor 2 mRNA to decrease toll-like receptor 2 protein and consequently suppresses P. acnes-induced proinflammatory cytokines.

PMID:
26902125
DOI:
10.1016/j.jid.2015.11.016
[Indexed for MEDLINE]
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