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Proc Natl Acad Sci U S A. 2015 Apr 21;112(16):E1969. doi: 10.1073/pnas.1503762112. Epub 2015 Mar 20.

Reply to Christ et al.: LQT1 and JLNS phenotypes in hiPSC-derived cardiomyocytes are due to KCNQ1 mutations.

Author information

1
Human Stem Cell Pluripotency Laboratory, Max Planck Institute for Molecular Biomedicine, D-48149 Münster, Germany; Chemical Genomics Centre of the Max Planck Society, 44227 Dortmund, Germany; boris.greber@mpi-muenster.mpg.de m.bellin@lumc.nl.
2
Department of Anatomy, Embryology, and Physiology, Heart Failure Research Center, Academic Medical Center, Amsterdam 1105 AZ, The Netherlands;
3
Department of Cardiovascular Medicine, Institute for Genetics of Heart Diseases, University Hospital Münster, D-48149 Münster, Germany; and.
4
Department of Anatomy and Embryology, Leiden University Medical Center, Leiden 2300 RC, The Netherlands.
5
Department of Anatomy and Embryology, Leiden University Medical Center, Leiden 2300 RC, The Netherlands boris.greber@mpi-muenster.mpg.de m.bellin@lumc.nl.
PMID:
25795241
PMCID:
PMC4413298
DOI:
10.1073/pnas.1503762112
[Indexed for MEDLINE]
Free PMC Article

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