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Cell. 2014 Dec 4;159(6):1247-9. doi: 10.1016/j.cell.2014.11.028.

Speciation via autoimmunity: a dangerous mix.

Author information

1
Department of Biology, University of Utah, Salt Lake City, UT 84112, USA. Electronic address: nitin.phadnis@utah.edu.
2
Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, WA 98109, USA; HHMI, Fred Hutchinson Cancer Research Center, Seattle, WA 98109, USA. Electronic address: hsmalik@fhcrc.org.

Abstract

In this issue of Cell, Chae et al. find that genomic "hot spots" encoding NLR plant immune receptor genes are recurrently responsible for hybrid necrosis, highlighting the role of host-pathogen evolutionary arms races in driving the evolution of hybrid incompatibilities.

PMID:
25480288
DOI:
10.1016/j.cell.2014.11.028
[Indexed for MEDLINE]
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