Unsuspected collision of synchronous lung adenocarcinomas: a potential cause of aberrant driver mutation profiles

J Thorac Oncol. 2014 Jan;9(1):e1-3. doi: 10.1097/JTO.0b013e3182a471c3.

Authors

Natasha Rekhtman¹, Laetitia Borsu, Boris Reva, Maria Arcila, Gregory J Riely, Marc Ladanyi, Alexander Drilon

Affiliation

¹ *Department of Pathology, †Human Oncology and Pathogenesis Program, Memorial Sloan-Kettering Cancer Center, New York, New York; ‡Computational Biology Program, Sloan-Kettering Institute, New York, New York; and §Department of Medicine Thoracic Oncology Service, Memorial Sloan-Kettering Cancer Center, New York, New York.

No abstract available

Publication types

Case Reports
Research Support, N.I.H., Extramural
Research Support, Non-U.S. Gov't

MeSH terms

Adenocarcinoma / genetics*
Adenocarcinoma of Lung
Aged
Female
Humans
Lung Neoplasms / genetics*
Mutation*
Neoplasms, Multiple Primary / genetics*
Proto-Oncogene Proteins / genetics*
Proto-Oncogene Proteins p21(ras)
ras Proteins / genetics*

Substances

KRAS protein, human
Proto-Oncogene Proteins
Proto-Oncogene Proteins p21(ras)
ras Proteins

Grants and funding

P01 CA129243/CA/NCI NIH HHS/United States