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Neuron. 2013 May 8;78(3):408-10. doi: 10.1016/j.neuron.2013.04.030.

Evidence for a common endocannabinoid-related pathomechanism in autism spectrum disorders.

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1
Department of Molecular Neurobiology, Max Planck Institute of Experimental Medicine, 37075 Göttingen, Germany. krueger@em.mpg.de

Abstract

In this issue of Neuron, Földy et al. (2013) report that endocannabinoid-mediated signaling at inhibitory synapses is dysregulated in mouse models of autism-associated Neuroligin-3 mutations. These findings carry implications regarding the pathophysiology of autism spectrum disorders and the development of treatment strategies.

PMID:
23664608
DOI:
10.1016/j.neuron.2013.04.030
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