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Neurosci Res. 2012 Sep;74(1):7-9. doi: 10.1016/j.neures.2012.06.001. Epub 2012 Jun 16.

Herpes zoster oticus: a clinical model for a transynaptic, reflex pathways, viral transmission hypotheses.

Author information

1
Otolaryngology Department, University Hospital of Modena, Via del Pozzo 71, 41100 Modena, Italy.

Abstract

Reactivation of the varicella-zoster virus (VZV) along the sensory nerves innervating the ear, including the geniculate ganglion, is responsible for herpes zoster oticus (HZO). In some cases, HZO is associated with polyneuropathy of the cranial nerves, although the mechanism of this involvement is not known. To explain this phenomenon and based on some clinical considerations, the present authors hypothesize an intersynaptic spread of VZV along the reflex pathways of the brainstem.

PMID:
22709534
DOI:
10.1016/j.neures.2012.06.001
[Indexed for MEDLINE]

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