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Items: 14

1.

Subtype selective NMDA receptor antagonists induce recovery of synapses lost following exposure to HIV-1 Tat.

Shin AH, Kim HJ, Thayer SA.

Br J Pharmacol. 2012 Jun;166(3):1002-17. doi: 10.1111/j.1476-5381.2011.01805.x.

2.

Neurotoxicity of human immunodeficiency virus-1: viral proteins and axonal transport.

Mocchetti I, Bachis A, Avdoshina V.

Neurotox Res. 2012 Jan;21(1):79-89. doi: 10.1007/s12640-011-9279-2. Review.

3.

Role of Tat protein in HIV neuropathogenesis.

Li W, Li G, Steiner J, Nath A.

Neurotox Res. 2009 Oct;16(3):205-20. doi: 10.1007/s12640-009-9047-8. Review.

PMID:
19526283
4.

HIV-tat induces formation of an LRP-PSD-95- NMDAR-nNOS complex that promotes apoptosis in neurons and astrocytes.

Eugenin EA, King JE, Nath A, Calderon TM, Zukin RS, Bennett MV, Berman JW.

Proc Natl Acad Sci U S A. 2007 Feb 27;104(9):3438-43.

5.

HIV tat and neurotoxicity.

King JE, Eugenin EA, Buckner CM, Berman JW.

Microbes Infect. 2006 Apr;8(5):1347-57. Review.

PMID:
16697675
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9.

Neurotoxic effects of the human immunodeficiency virus type-1 transcription factor Tat require function of a polyamine sensitive-site on the N-methyl-D-aspartate receptor.

Prendergast MA, Rogers DT, Mulholland PJ, Littleton JM, Wilkins LH Jr, Self RL, Nath A.

Brain Res. 2002 Nov 8;954(2):300-7.

PMID:
12414113
10.

HIV-1 Tat causes apoptotic death and calcium homeostasis alterations in rat neurons.

Bonavia R, Bajetto A, Barbero S, Albini A, Noonan DM, Schettini G.

Biochem Biophys Res Commun. 2001 Oct 26;288(2):301-8.

PMID:
11606043
11.

HIV-1 Tat through phosphorylation of NMDA receptors potentiates glutamate excitotoxicity.

Haughey NJ, Nath A, Mattson MP, Slevin JT, Geiger JD.

J Neurochem. 2001 Aug;78(3):457-67.

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Human immunodeficiency virus type 1 tat activates non-N-methyl-D-aspartate excitatory amino acid receptors and causes neurotoxicity.

Magnuson DS, Knudsen BE, Geiger JD, Brownstone RM, Nath A.

Ann Neurol. 1995 Mar;37(3):373-80.

PMID:
7695237

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