Format

Send to

Choose Destination
Med Sci Sports Exerc. 2012 Mar;44(3):534-41. doi: 10.1249/MSS.0b013e318232cf77.

Effects of deception on exercise performance: implications for determinants of fatigue in humans.

Author information

1
School of Life Sciences, Northumbria University, Newcastle Upon Tyne, United Kingdom. mark.stone@northumbria.ac.uk

Abstract

PURPOSE:

The aim of this study was to investigate whether it was possible to reduce the time taken to complete a 4000-m cycling time trial by misleading participants into believing they were racing against a previous trial, when, in fact, the power output was 2% greater.

METHODS:

Nine trained male cyclists each completed four 4000-m time trials. The first trial was a habituation and the data from the second trial was used to form a baseline (BL). During trials 3 and 4, participants raced against an avatar, which they were informed represented their BL performance. However, whereas one of these trials was an accurate (ACC) representation of BL, the power output in the other trial was set at 102% of BL and formed the deception condition (DEC). Oxygen uptake and RER were measured continuously and used to determine aerobic and anaerobic contributions to power output.

RESULTS:

There was a significant difference between trials for time to completion (F = 15.3, P = 0.00). Participants completed DEC more quickly than BL (90% CI = 2.1-10.1 s) and ACC (90% CI = 1.5-5.4 s) and completed ACC more quickly than BL (90% CI = 0.5-4.8 s). The difference in performance between DEC and ACC was attributable to a greater anaerobic contribution to power output at 90% of the total distance (F = 5.3, P = 0.02, 90% CI = 4-37 W).

CONCLUSIONS:

The provision of surreptitiously augmented feedback derived from a previous performance reduces time taken for cyclists to accomplish a time trial of known duration. This suggests that cyclists operate with a metabolic reserve even during maximal time trials and that this reserve can be accessed after deception.

PMID:
21886012
DOI:
10.1249/MSS.0b013e318232cf77
[Indexed for MEDLINE]

Supplemental Content

Full text links

Icon for Wolters Kluwer
Loading ...
Support Center