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Electromagn Biol Med. 2009;28(1):53-60. doi: 10.1080/15368370802711805.

Cancer growth and its inhibition in terms of coherence.

Author information

1
International Institute of Biophysics, Neuss, Germany. iib@lifescientists.de

Abstract

It is shown that a molecular origin for growth inhibition is rather unlikely because the cross-sectional area of inhibitory forces in a cell population cannot exceed more than about 10(-8) Dalton. A model of the time dependence of cell number N(t), where t is the time, is based on biophotons and explains without any contradiction to known experimental results growth regulation in terms of the factor a = 1/T, which stimulates the cell division rate dN/dt and the factor b = dT/dN(1/T(2)), which inhibits cell division. It accounts for the total cell division rate dN/dt = aN(t) - bN(2)(t). For adults, T is the coherence time of about 10(6) s, corresponding to the longest lifetime of cell organelles in men, while dT/dN = 10(-7) s corresponds to the resolution time of the cell population which is always the average time interval between two cell loss events. Our model follows a stringently holistic approach to describing a cell population as an entity, regulated by a fully coherent (biophoton) field.

PMID:
19337895
DOI:
10.1080/15368370802711805
[Indexed for MEDLINE]

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