Format

Send to

Choose Destination
See comment in PubMed Commons below
Oral Surg Oral Med Oral Pathol Oral Radiol Endod. 2009 Apr;107(4):493-8. doi: 10.1016/j.tripleo.2008.10.016. Epub 2009 Jan 4.

Traumatic intrusion of primary teeth and its effects on the permanent successors: a clinical follow-up study.

Author information

1
Department of Pediatric Dentistry, Center of Dental Sciences, Gülhane Medical Academy, Ankara, Turkey. ceyhanaltun@yahoo.com

Abstract

OBJECTIVE:

Owing to the anatomic proximity between the root of the primary tooth and its permanent successor, trauma to primary dentition may cause developmental disturbances in unerupted permanent teeth.

STUDY DESIGN:

This report presents the findings of a prospective 7-year study examining developmental disturbances of permanent teeth attributed to intrusive injury of their primary predecessors based on initial patient records and clinical observation. Intrusive injuries were diagnosed and grouped according to World Health Organization classifications, as modified by Andreasen et al.

RESULTS:

A total of 78 children (aged 12-48 months) presenting with intrusive trauma to 138 primary incisors were available for follow-up examinations. The most frequently intruded primary teeth were maxillary incisors (93.47%), with the right central primary incisor representing 41.3% of all intruded teeth. In 23 patients, 36 teeth were scheduled for extraction during the first visit. During the course of follow-up, 41 of the remaining teeth exhibited post-traumatic consequences, including pulp necrosis (78.0%), internal and/or external root resorption (14.6%), ankylosis (4.9%), and obliteration of the pulp canal (2.5%). In 74 permanent successors (53.6%), >or=1 of the following developmental disturbances were observed: enamel hypoplasia (28.3%), crown and/or root deformation (16.7%), and ectopic eruption (16.7%).

CONCLUSION:

There was no significant correlation between age of intrusion and frequency of subsequent developmental disturbances (P > .05).

PMID:
19121957
DOI:
10.1016/j.tripleo.2008.10.016
[Indexed for MEDLINE]
PubMed Commons home

PubMed Commons

0 comments
How to join PubMed Commons

    Supplemental Content

    Full text links

    Icon for Elsevier Science
    Loading ...
    Support Center