Abstract
What are the pathways that underlie the coordinated responses of an organism to well-fed and food-deprived states? A report in this issue of Cell Metabolism suggests that starvation functions via a muscarinic acetylcholine receptor to activate MAP kinase signaling in the pharyngeal muscle of C. elegans (You et al., 2006).
MeSH terms
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Acetylcholine / physiology
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Animals
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Arecoline / pharmacology
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Caenorhabditis elegans / genetics
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Caenorhabditis elegans / physiology*
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Caenorhabditis elegans Proteins / genetics
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Caenorhabditis elegans Proteins / physiology*
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Cholinergic Agonists / pharmacology
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Enzyme Activation / drug effects
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Feeding Behavior
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GTP-Binding Protein beta Subunits / genetics
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GTP-Binding Protein beta Subunits / physiology
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MAP Kinase Signaling System*
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Mitogen-Activated Protein Kinases / metabolism*
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Muscarinic Antagonists / pharmacology
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Mutation
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Pharynx / drug effects
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Pharynx / enzymology
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Receptors, Muscarinic / physiology*
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Starvation
Substances
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Caenorhabditis elegans Proteins
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Cholinergic Agonists
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GPB-2 protein, C elegans
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GTP-Binding Protein beta Subunits
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Muscarinic Antagonists
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Receptors, Muscarinic
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Arecoline
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Mitogen-Activated Protein Kinases
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Acetylcholine