Mapping out starvation responses

Cell Metab. 2006 Apr;3(4):235-6. doi: 10.1016/j.cmet.2006.03.002.

Abstract

What are the pathways that underlie the coordinated responses of an organism to well-fed and food-deprived states? A report in this issue of Cell Metabolism suggests that starvation functions via a muscarinic acetylcholine receptor to activate MAP kinase signaling in the pharyngeal muscle of C. elegans (You et al., 2006).

Publication types

  • Comment

MeSH terms

  • Acetylcholine / physiology
  • Animals
  • Arecoline / pharmacology
  • Caenorhabditis elegans / genetics
  • Caenorhabditis elegans / physiology*
  • Caenorhabditis elegans Proteins / genetics
  • Caenorhabditis elegans Proteins / physiology*
  • Cholinergic Agonists / pharmacology
  • Enzyme Activation / drug effects
  • Feeding Behavior
  • GTP-Binding Protein beta Subunits / genetics
  • GTP-Binding Protein beta Subunits / physiology
  • MAP Kinase Signaling System*
  • Mitogen-Activated Protein Kinases / metabolism*
  • Muscarinic Antagonists / pharmacology
  • Mutation
  • Pharynx / drug effects
  • Pharynx / enzymology
  • Receptors, Muscarinic / physiology*
  • Starvation

Substances

  • Caenorhabditis elegans Proteins
  • Cholinergic Agonists
  • GPB-2 protein, C elegans
  • GTP-Binding Protein beta Subunits
  • Muscarinic Antagonists
  • Receptors, Muscarinic
  • Arecoline
  • Mitogen-Activated Protein Kinases
  • Acetylcholine