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Arch Neurol. 2004 Apr;61(4):583-5.

Rubral lateropulsion due to vertebral artery dissection in a patient with Klippel-Feil syndrome.

Author information

1
Department of Neurology, University of Iowa Hospitals and Clinics, Iowa City, USA. morvaridkarimi@hotmail.com

Abstract

BACKGROUND:

Neurologic deficits in patients with Klippel-Feil syndrome usually are attributed to direct compression of neuronal structures or hypoperfusion secondary to compression of the vertebral arteries by bony abnormalities.

OBJECTIVE:

To describe a 38-year-old woman with known Klippel-Feil syndrome who developed lateropulsion.

RESULTS:

The results of magnetic resonance imaging were consistent with rubrothalamic stroke. The cerebral angiogram confirmed vertebral artery dissection at the level of her previously observed bony abnormality.

CONCLUSIONS:

Hypermobility adjacent to fused vertebrae subjects the vertebral artery to increased shear forces. Thus, Klippel-Feil syndrome may be a predisposing factor for vertebral artery dissection. Moreover, to our knowledge, this case represents the second known case of rubral lateropulsion.

PMID:
15096409
DOI:
10.1001/archneur.61.4.583
[Indexed for MEDLINE]
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