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Fig. 6. MicroRNA-708 (miR-708) inhibits the AKT/β-catenin pathway.. From: Silencing of microRNA-708 promotes cell growth and epithelial-to-mesenchymal transition by activating the SPHK2/AKT/β-catenin pathway in glioma.
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Fig. 1. The level of microRNA-708 (miR-708) expression was down-regulated in glioma cell lines and tissues.. From: Silencing of microRNA-708 promotes cell growth and epithelial-to-mesenchymal transition by activating the SPHK2/AKT/β-catenin pathway in glioma.
Fig. 4. MicroRNA-708 (miR-708) directly targeted sphingosine kinase 2 (SPHK2) in glioma cells.. From: Silencing of microRNA-708 promotes cell growth and epithelial-to-mesenchymal transition by activating the SPHK2/AKT/β-catenin pathway in glioma.
Fig. 7. Enhancer of zeste homolog 2 (EZH2) inhibited microRNA-708 (miR-708) expression.. From: Silencing of microRNA-708 promotes cell growth and epithelial-to-mesenchymal transition by activating the SPHK2/AKT/β-catenin pathway in glioma.
Fig. 5. Sphingosine kinase 2 (SPHK2) was a glioma oncogene.. From: Silencing of microRNA-708 promotes cell growth and epithelial-to-mesenchymal transition by activating the SPHK2/AKT/β-catenin pathway in glioma.
Fig. 3. Overexpression of microRNA-708 (miR-708) decreased glioma cell invasion through reversing the epithelial-to-mesenchymal transition (EMT) phenotype.. From: Silencing of microRNA-708 promotes cell growth and epithelial-to-mesenchymal transition by activating the SPHK2/AKT/β-catenin pathway in glioma.
Fig. 8. microRNA-708 (miR-708 predicted poor glioma patient prognosis and was negatively associated with enhancer of zeste homolog 2 (EZH2) expression.. From: Silencing of microRNA-708 promotes cell growth and epithelial-to-mesenchymal transition by activating the SPHK2/AKT/β-catenin pathway in glioma.
Fig. 2. Overexpression of microRNA-708 (miR-708) decreased glioma cell growth both in vitro and in vivo.. From: Silencing of microRNA-708 promotes cell growth and epithelial-to-mesenchymal transition by activating the SPHK2/AKT/β-catenin pathway in glioma.
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