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Fig 1. Panobinostat causes increased growth delay in irradiated bladder cancer cell xenografts.. From: Radiosensitisation in vivo by histone deacetylase inhibition with no increase in early normal tissue radiation toxicity.
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Fig 5. HDAC expression in response to panobinostat in a panel of bladder cell lines.. From: Radiosensitisation in vivo by histone deacetylase inhibition with no increase in early normal tissue radiation toxicity.
Fig 6. Effects of class I HDAC inhibitors on bladder cancer cells.. From: Radiosensitisation in vivo by histone deacetylase inhibition with no increase in early normal tissue radiation toxicity.
Fig 4. Panobinostat causes no increased radiation normal tissue toxicity at 11-12 weeks.. From: Radiosensitisation in vivo by histone deacetylase inhibition with no increase in early normal tissue radiation toxicity.
Fig 3. Methodology to assess acute toxicity using the small animal radiation research platform.. From: Radiosensitisation in vivo by histone deacetylase inhibition with no increase in early normal tissue radiation toxicity.
Fig 2. Panobinostat accumulation in normal tissues and methodology to assess acute toxicity.. From: Radiosensitisation in vivo by histone deacetylase inhibition with no increase in early normal tissue radiation toxicity.
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