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Figure 5. MD Simulations of the EGFR Regulatory Dimers.. From: Computational Modeling of Allosteric Communication Reveals Organizing Principles of Mutation-Induced Signaling in ABL and EGFR Kinases.
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Figure 2. Structural Mapping of Allosteric Communication Profiles in the ABL Regulatory Complexes.. From: Computational Modeling of Allosteric Communication Reveals Organizing Principles of Mutation-Induced Signaling in ABL and EGFR Kinases.
Figure 8. Structural Analysis of the Inter-monomer Interface in Functional Asymmetric EGFR Dimer.. From: Computational Modeling of Allosteric Communication Reveals Organizing Principles of Mutation-Induced Signaling in ABL and EGFR Kinases.
Figure 9. Structural Analysis of the Inter-monomer Interface in a Symmetric EGFR Dimer.. From: Computational Modeling of Allosteric Communication Reveals Organizing Principles of Mutation-Induced Signaling in ABL and EGFR Kinases.
Figure 3. Structure-Functional Analysis of the Inter-Domain Interface in the ABL Regulatory Complexes.. From: Computational Modeling of Allosteric Communication Reveals Organizing Principles of Mutation-Induced Signaling in ABL and EGFR Kinases.
Figure 4. Structural Mapping of Allosteric Communication Profiles in the EGFR Kinase Catalytic Domain.. From: Computational Modeling of Allosteric Communication Reveals Organizing Principles of Mutation-Induced Signaling in ABL and EGFR Kinases.
Figure 7. Mutation-Induced Allosteric Signaling in the EGFR Kinase Regulatory Dimers: Analysis of Long-Range Communications between EGFR Monomers.. From: Computational Modeling of Allosteric Communication Reveals Organizing Principles of Mutation-Induced Signaling in ABL and EGFR Kinases.
Figure 1. Structural Mapping of Allosteric Communication Profiles in the ABL Kinase Catalytic Domain.. From: Computational Modeling of Allosteric Communication Reveals Organizing Principles of Mutation-Induced Signaling in ABL and EGFR Kinases.
Figure 6. Allosteric signaling in the EGFR Kinase Regulatory Dimers: The Intra-monomer Communications.. From: Computational Modeling of Allosteric Communication Reveals Organizing Principles of Mutation-Induced Signaling in ABL and EGFR Kinases.
Figure 10. Allosteric Communications and Stabilizing Interactions in an Extended Structure of a Symmetric EGFR Dimer.. From: Computational Modeling of Allosteric Communication Reveals Organizing Principles of Mutation-Induced Signaling in ABL and EGFR Kinases.
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