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1.
Figure 2

Figure 2. Schematic representation of various transcription factors that are modulated by reactive oxygen species. From: Oxidative stress, inflammation, and cancer: How are they linked?.

Simone Reuter, et al. Free Radic Biol Med. ;49(11):1603-1616.

CitationFull text

2.
Figure 1

Figure 1. Schematic representation of various activators and inhibitors of reactive oxygen species production. From: Oxidative stress, inflammation, and cancer: How are they linked?.

Simone Reuter, et al. Free Radic Biol Med. ;49(11):1603-1616.

CitationFull text

3.
Figure 3

Figure 3. Model of a balance between pro-oxidants and anti-oxidants. From: Oxidative stress, inflammation, and cancer: How are they linked?.

Under normal conditions, anti-oxidants outbalance pro-oxidants, but under oxidative conditions, pro-oxidants prevail over anti-oxidants, which can lead to many inflammatory diseases including cancer.

Simone Reuter, et al. Free Radic Biol Med. ;49(11):1603-1616.

CitationFull text

4.
Figure 4

Figure 4. Model of the sensitivity of normal cells versus cancer cells to reactive oxygen species. From: Oxidative stress, inflammation, and cancer: How are they linked?.

Normal cells are hypersensitive to ROS if not adequately protected by anti-oxidant mechanisms, which may lead to cancer formation. Cancer cells, on the other hand, have upregulated antioxidant mechanisms (glutathione, SOD, catalase, and others) that will protect them against ROS, as can be observed in, for example, the case of radioresistance.

Simone Reuter, et al. Free Radic Biol Med. ;49(11):1603-1616.

CitationFull text

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