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Figure 1. Topology of the hERG1 Markov model.. From: Differential Expression of hERG1 Channel Isoforms Reproduces Properties of Native I Kr and Modulates Cardiac Action Potential Characteristics.
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Figure 6. Restitution slope is modulated by the relative abundance of hERG1a and hERG1b.. From: Differential Expression of hERG1 Channel Isoforms Reproduces Properties of Native I Kr and Modulates Cardiac Action Potential Characteristics.
Figure 2. The relative abundance of hERG1a and hERG1b determines the ‘peak potential’ in mammalian cells.. From: Differential Expression of hERG1 Channel Isoforms Reproduces Properties of Native I Kr and Modulates Cardiac Action Potential Characteristics.
Figure 3. The ‘peak potential’ is directly dependent on the relative abundance of hERG1 isoforms.. From: Differential Expression of hERG1 Channel Isoforms Reproduces Properties of Native I Kr and Modulates Cardiac Action Potential Characteristics.
Figure 5. Action potential duration is modulated by the relative abundance of hERG1a and hERG1b.. From: Differential Expression of hERG1 Channel Isoforms Reproduces Properties of Native I Kr and Modulates Cardiac Action Potential Characteristics.
Figure 4. The experimental data is reproduced by a hERG1 Markov model.. From: Differential Expression of hERG1 Channel Isoforms Reproduces Properties of Native I Kr and Modulates Cardiac Action Potential Characteristics.
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