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Figure 2

Figure 2. From: Epidermal Growth Factor Receptor (EGFR) High Gene Copy Number and Activating Mutations in Lung Adenocarcinomas Are Not Consistently Accompanied by Positivity for EGFR Protein by Standard Immunohistochemistry.

EGFR protein expression of the samples with different genotypes. The expression of EGFR protein was determined by immunohistochemistry using Dako EGFR PharmDx kits (DakoCytomation), and the slides were counterstained with hematoxylin. *High polysomy, not amplified.

Ferenc Pinter, et al. J Mol Diagn. 2008 Mar;10(2):160-168.

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2.
Figure 3

Figure 3. From: Epidermal Growth Factor Receptor (EGFR) High Gene Copy Number and Activating Mutations in Lung Adenocarcinomas Are Not Consistently Accompanied by Positivity for EGFR Protein by Standard Immunohistochemistry.

Subgroups of lung adenocarcinomas designated by the three EGFR status analyses. Since the population of this three-sided analysis has been only a subgroup of the population of the two-sided analyses, the analogous frequencies are slightly different (mutation+, 15.6%; FISH+, 42.2%; amplification+, 6.7%; IHC+, 60%). AMP, amplification; freq: frequency; MUT, mutation. *Subgroup of the FISH+ group.

Ferenc Pinter, et al. J Mol Diagn. 2008 Mar;10(2):160-168.

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3.
Figure 1

Figure 1. From: Epidermal Growth Factor Receptor (EGFR) High Gene Copy Number and Activating Mutations in Lung Adenocarcinomas Are Not Consistently Accompanied by Positivity for EGFR Protein by Standard Immunohistochemistry.

Chromatogram of the del2235-2249>del746-750 exon 19 deletion (A) and T2573G>L858R exon 21 point mutation (B). FISH analysis of disomy (C), high trisomy (D), high polysomy (E), and gene amplification (F). The EGFR gene signal is red; the chromosome 7 signal is green.

Ferenc Pinter, et al. J Mol Diagn. 2008 Mar;10(2):160-168.

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