|
| Status |
Public on May 10, 2016 |
| Title |
Nfib promotes Metastasis through a Widespread Increase in Chromatin Accessibility [RNA-seq] |
| Organism |
Mus musculus |
| Experiment type |
Expression profiling by high throughput sequencing
|
| Summary |
We describe Nfib as an important regulator of chromatin accessibility in SCLC.
|
| |
|
| Overall design |
We performed RNA-seq on three cell lines with either knockdown of Nfib expression or overexpression of Nfib.
Cell lines 16T and KP1 were expressing iether a control vector or a vector expressing shRNA to Nfib, in duplicate
Cell line KP22 were expressing either an empty vector or a vector expressing Nfib cDNA, in duplicate.
|
| |
|
| Contributor(s) |
Denny S, Yang D, Sage J, Greenleaf W, Winslow M |
| Citation(s) |
27374332 |
| |
| Submission date |
May 09, 2016 |
| Last update date |
May 15, 2019 |
| Contact name |
William Greenleaf |
| E-mail(s) |
wjg@stanford.edu
|
| Organization name |
Stanford University
|
| Street address |
279 Campus Drive
|
| City |
Stanford |
| State/province |
CA |
| ZIP/Postal code |
94305 |
| Country |
USA |
| |
|
| Platforms (1) |
| GPL17021 |
Illumina HiSeq 2500 (Mus musculus) |
|
| Samples (12)
|
|
| This SubSeries is part of SuperSeries: |
| GSE81258 |
Nfib promotes Metastasis through a Widespread Increase in Chromatin Accessibility |
|
| Relations |
| BioProject |
PRJNA321071 |
| SRA |
SRP074735 |
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