|
Status |
Public on May 10, 2016 |
Title |
Nfib promotes Metastasis through a Widespread Increase in Chromatin Accessibility [ChIP-seq] |
Organism |
Mus musculus |
Experiment type |
Genome binding/occupancy profiling by high throughput sequencing
|
Summary |
We describe Nfib as an important regulator of chromatin accessibility in SCLC.
|
|
|
Overall design |
We performed ChIP seq for Nfib in three different cell lines. Replicates of input and pulldowns for three cell lines is given.
|
|
|
Contributor(s) |
Denny S, Yang D, Sage J, Greenleaf W, Winslow M |
Citation(s) |
27374332 |
|
Submission date |
May 09, 2016 |
Last update date |
May 15, 2019 |
Contact name |
William Greenleaf |
E-mail(s) |
wjg@stanford.edu
|
Organization name |
Stanford University
|
Street address |
279 Campus Drive
|
City |
Stanford |
State/province |
CA |
ZIP/Postal code |
94305 |
Country |
USA |
|
|
Platforms (1) |
GPL17021 |
Illumina HiSeq 2500 (Mus musculus) |
|
Samples (12)
|
|
This SubSeries is part of SuperSeries: |
GSE81258 |
Nfib promotes Metastasis through a Widespread Increase in Chromatin Accessibility |
|
Relations |
BioProject |
PRJNA321072 |
SRA |
SRP074734 |